Erika C Crouch
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Explore the profile of Erika C Crouch including associated specialties, affiliations and a list of published articles.
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63
Citations
1630
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Recent Articles
1.
Wu K, Zhang Y, Yin-DeClue H, Sun K, Mao D, Crouch E, et al.
medRxiv
. 2024 Sep;
PMID: 39252892
Epithelial injury calls for a regenerative response from a coordinated network of epithelial stem cells and immune cells. Defining this network is key to preserving the repair process for acute...
2.
Zhang Y, Wu K, Mao D, Iberg C, Yin-DeClue H, Sun K, et al.
bioRxiv
. 2024 Sep;
PMID: 39229202
The stress kinase MAPK13 (aka p38δ-MAPK) is an attractive entry point for therapeutic intervention because it regulates the structural remodeling that can develop after epithelial barrier injury in the lung...
3.
Wu K, Zhang Y, Yin-DeClue H, Sun K, Mao D, Yang K, et al.
J Clin Invest
. 2024 Jul;
134(18).
PMID: 39052353
Epithelial barriers are programmed for defense and repair but are also the site of long-term structural remodeling and disease. In general, this paradigm features epithelial stem cells (ESCs) that are...
4.
Wu K, Zhang Y, Mao D, Iberg C, Yin-DeClue H, Sun K, et al.
bioRxiv
. 2024 Jun;
PMID: 38895360
All living organisms are charged with repair after injury particularly at epithelial barrier sites, but in some cases this response leads instead to structural remodeling and long-term disease. Identifying the...
5.
Wu K, Zhang Y, Austin S, Yin-DeClue H, Byers D, Crouch E, et al.
Am J Pathol
. 2023 Mar;
193(6):680-689.
PMID: 36868468
Respiratory viruses, including severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), can trigger chronic lung disease that persists and even progresses after expected clearance of infectious virus. To gain an understanding...
6.
Wu K, Zhang Y, Austin S, Declue H, Byers D, Crouch E, et al.
medRxiv
. 2022 Sep;
PMID: 36172126
Respiratory viruses, including SARS-CoV-2, can trigger chronic lung disease that persists and even progresses after expected clearance of infectious virus. To gain an understanding of this process, we examined a...
7.
White M, Nikolaidis N, McCormack F, Crouch E, Hartshorn K
Front Microbiol
. 2021 Dec;
12:774711.
PMID: 34956139
Mannose-binding lectins effectively inhibit most seasonal strains of influenza A virus and contribute to the innate host defense vs. these viruses. In contrast, pandemic IAV strains are largely resistant to...
8.
Wu K, Kamimoto K, Zhang Y, Yang K, Keeler S, Gerovac B, et al.
J Clin Invest
. 2021 Aug;
131(19).
PMID: 34343135
Epithelial cells are charged with protection at barrier sites, but whether this normally beneficial response might sometimes become dysfunctional still needs definition. Here, we recognized a pattern of imbalance marked...
9.
White M, Tripathi S, Verma A, Kingma P, Takahashi K, Jensenius J, et al.
Innate Immun
. 2016 Nov;
23(1):77-88.
PMID: 27856789
Infiltrating activated monocytes are important mediators of damaging inflammation during influenza A virus (IAV) infection. We show that soluble respiratory proteins [collectins, surfactant proteins D (SP-D) and mannose binding lectin...
10.
Reinhardt A, Wehle M, Geissner A, Crouch E, Kang Y, Yang Y, et al.
J Struct Biol
. 2016 Jun;
195(3):387-395.
PMID: 27350640
As a major player of the innate immune system, surfactant protein D (SP-D) recognizes and promotes elimination of various pathogens such as Gram-negative bacteria. SP-D binds to l-glycero-d-manno-heptose (Hep), a...