David M Tarlinton
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Explore the profile of David M Tarlinton including associated specialties, affiliations and a list of published articles.
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115
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6514
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Recent Articles
21.
Kara E, Bastow C, McKenzie D, Gregor C, Fenix K, Babb R, et al.
J Exp Med
. 2018 Feb;
215(3):801-813.
PMID: 29386231
Activated B cells can initially differentiate into three functionally distinct fates-early plasmablasts (PBs), germinal center (GC) B cells, or early memory B cells-by mechanisms that remain poorly understood. Here, we...
22.
Willis S, Tellier J, Liao Y, Trezise S, Light A, ODonnell K, et al.
Nat Commun
. 2017 Nov;
8(1):1426.
PMID: 29127283
Humoral immunity requires B cells to respond to multiple stimuli, including antigen, membrane and soluble ligands, and microbial products. Ets family transcription factors regulate many aspects of haematopoiesis, although their...
23.
King A, Li L, Wong D, Liu R, Bamford R, Strasser A, et al.
PLoS Genet
. 2017 Sep;
13(9):e1007010.
PMID: 28922373
Mechanistic differences in the development and function of adaptive, high-affinity antibody-producing B-2 cells and innate-like, "natural" antibody-producing B-1a cells remain poorly understood. Here we show that the multi-functional dynein light...
24.
Chevrier S, Kratina T, Emslie D, Tarlinton D, Corcoran L
Immunol Cell Biol
. 2017 Sep;
95(10):925-932.
PMID: 28875978
Bcl6 (B-cell lymphoma 6) is a transcriptional repressor and critical mediator of the germinal center reaction during a T-cell-dependent antibody response, where it enables somatic hypermutation of immunoglobulin genes and...
25.
Carrington E, Tarlinton D, Gray D, Huntington N, Zhan Y, Lew A
Immunol Cell Biol
. 2017 Sep;
95(10):870-877.
PMID: 28875977
Targeting survival mechanisms of immune cells may provide an avenue for immune intervention to dampen unwanted responses (e.g. autoimmunity, immunopathology and transplant rejection) or enhance beneficial ones (e.g. immune deficiency,...
26.
Dolezal E, Infantino S, Drepper F, Borsig T, Singh A, Wossning T, et al.
Nat Immunol
. 2017 Jun;
18(8):911-920.
PMID: 28628091
Developing pre-B cells in the bone marrow alternate between proliferation and differentiation phases. We found that protein arginine methyl transferase 1 (PRMT1) and B cell translocation gene 2 (BTG2) are...
27.
Piovesan D, Tempany J, Di Pietro A, Baas I, Yiannis C, ODonnell K, et al.
Cell Rep
. 2017 Apr;
19(3):461-470.
PMID: 28423310
Humoral immune responses are tailored to the invading pathogen through regulation of key transcription factors and their networks. This is critical to establishing effective antibody-mediated responses, yet it is unknown...
28.
Carrington E, Zhan Y, Brady J, Zhang J, Sutherland R, Anstee N, et al.
Cell Death Differ
. 2017 Apr;
24(5):878-888.
PMID: 28362427
Survival of various immune cell populations has been proposed to preferentially rely on a particular anti-apoptotic BCL-2 family member, for example, naive T cells require BCL-2, while regulatory T cells...
29.
Vikstrom I, Slomp A, Carrington E, Moesbergen L, Chang C, Kelly G, et al.
Cell Death Dis
. 2016 Aug;
7(8):e2345.
PMID: 27560714
Pro-survival BCL-2 family members protect cells from programmed cell death that can be induced by multiple internal or external cues. Within the haematopoietic lineages, the BCL-2 family members BCL-2, BCL-XL...
30.
Chow K, Delconte R, Huntington N, Tarlinton D, Sutherland R, Zhan Y, et al.
J Immunol
. 2016 Jul;
197(5):2000-8.
PMID: 27474076
Although the mechanisms governing the innate recognition of pathogen-associated molecular patterns have been well defined, how allogeneic cellular stimuli evoke innate responses remains less so. In this article, we report...