Brian L Lin
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Explore the profile of Brian L Lin including associated specialties, affiliations and a list of published articles.
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13
Citations
143
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Recent Articles
1.
Taube N, Steiner M, Ebenebe-Kasonde O, Kabir R, Garbus-Grant H, Alam El Din S, et al.
Am J Physiol Heart Circ Physiol
. 2025 Jan;
328(3):H460-H471.
PMID: 39888327
Cardiovascular disease is the leading cause of mortality in the United States. Studies suggest a role for environmental exposures in the etiology of cardiovascular disease, including exposure to arsenic through...
2.
Taube N, Steiner M, Ebenebe-Kasonde O, Kabir R, Garbus-Grant H, Alam El Din S, et al.
bioRxiv
. 2024 Oct;
PMID: 39386735
New & Noteworthy: Gestational exposure to sodium arsenite at environmentally relevant doses (100 and 1000 µg/L) increases postpartum heart size, and induces dysregulated Ca homeostasis and impaired shortening in isolated...
3.
Hart C, Lee Y, Xie J, Gao G, Lin B, Hammers D, et al.
JCI Insight
. 2024 May;
9(11).
PMID: 38713520
Clinical trials delivering high doses of adeno-associated viruses (AAVs) expressing truncated dystrophin molecules (microdystrophins) are underway for Duchenne muscular dystrophy (DMD). We examined the efficiency and efficacy of this strategy...
4.
Reyes Gaido O, Pavlaki N, Granger J, Mesubi O, Liu B, Lin B, et al.
Sci Transl Med
. 2023 Jun;
15(701):eabq7839.
PMID: 37343080
Ca/calmodulin-dependent protein kinase II (CaMKII) hyperactivity causes cardiac arrhythmias, a major source of morbidity and mortality worldwide. Despite proven benefits of CaMKII inhibition in numerous preclinical models of heart disease,...
5.
Fitch M, Kabir R, Ebenebe O, Taube N, Garbus H, Sinha P, et al.
Life Sci
. 2023 Apr;
324:121712.
PMID: 37100378
Aims: Cadmium exposure is a worldwide problem that has been linked to the development of cardiovascular disease. This study aimed to elucidate mechanistic details of chronic cadmium exposure on the...
6.
Lin B, Shin J, Jeffreys W, Wang N, Lukban C, Moorer M, et al.
JCI Insight
. 2022 Sep;
7(19).
PMID: 36099033
Gene mutations causing loss of dystrophin result in the severe muscle disease known as Duchenne muscular dystrophy (DMD). Despite efforts at genetic repair, DMD therapy remains largely palliative. Loss of...
7.
Boyer J, Huo J, Han S, Havens J, Prasad V, Lin B, et al.
Nat Commun
. 2022 May;
13(1):2940.
PMID: 35618700
Skeletal muscle can repair and regenerate due to resident stem cells known as satellite cells. The muscular dystrophies are progressive muscle wasting diseases underscored by chronic muscle damage that is...
8.
Kannan S, Farid M, Lin B, Miyamoto M, Kwon C
PLoS Comput Biol
. 2021 Sep;
17(9):e1009305.
PMID: 34534204
The immaturity of pluripotent stem cell (PSC)-derived tissues has emerged as a universal problem for their biomedical applications. While efforts have been made to generate adult-like cells from PSCs, direct...
9.
Verma R, Kannan S, Lin B, Fomchenko K, Nieuwenhuis T, Patil A, et al.
Skelet Muscle
. 2021 May;
11(1):13.
PMID: 34001262
Background: Skeletal muscle myofibers can be separated into functionally distinct cell types that differ in gene and protein expression. Current single cell expression data is generally based upon single nucleus...
10.
Chan X, Volkova E, Eoh J, Black R, Fang L, Gorashi R, et al.
iScience
. 2021 Apr;
24(4):102246.
PMID: 33796838
Heterozygous gain-of-function (GOF) mutations of hypoxia-inducible factor 2α (HIF2A), a key hypoxia-sensing regulator, are associated with erythrocytosis, thrombosis, and vascular complications that account for morbidity and mortality of patients. We...