Andrew L Koenig
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Explore the profile of Andrew L Koenig including associated specialties, affiliations and a list of published articles.
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18
Citations
653
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Recent Articles
1.
Terada Y, Li W, Amrute J, Bery A, Liu C, Nunna V, et al.
Cell Rep
. 2025 Mar;
44(3):115380.
PMID: 40042972
Triggering receptor expressed on myeloid cells 1 (TREM-1) has been shown to amplify inflammatory signals, such as Toll-like receptor signaling, after infection and sterile injury. While previous studies have demonstrated...
2.
Koenig A, Kadyrov F, Amrute J, Yang S, Weinheimer C, Nigro J, et al.
bioRxiv
. 2025 Feb;
PMID: 39974922
Inflammation contributes to the pathogenesis of myocardial infarction and heart failure and represents a viable therapeutic target. Monocytes and their progeny are highly abundant and display incredible functional diversity, serving...
3.
Amrute J, Luo X, Penna V, Yang S, Yamawaki T, Hayat S, et al.
Nature
. 2024 Oct;
635(8038):423-433.
PMID: 39443792
Inflammation and tissue fibrosis co-exist and are causally linked to organ dysfunction. However, the molecular mechanisms driving immune-fibroblast cell communication in human cardiac disease remain unexplored and there are at...
4.
Kadyrov F, Koenig A, Amrute J, Dun H, Li W, Weinheimer C, et al.
Nat Cardiovasc Res
. 2024 Oct;
3(11):1337-1355.
PMID: 39433910
Myocardial infarction initiates cardiac remodeling and is central to heart failure pathogenesis. Following myocardial ischemia-reperfusion injury, monocytes enter the heart and differentiate into diverse subpopulations of macrophages. Here we show...
5.
de Winter N, Ji J, Sintou A, Forte E, Lee M, Noseda M, et al.
J Mol Cell Cardiol
. 2024 May;
192:48-64.
PMID: 38734060
Introduction: Chronic immunopathology contributes to the development of heart failure after a myocardial infarction. Both T and B cells of the adaptive immune system are present in the myocardium and...
6.
Leid J, Gray R, Rakita P, Koenig A, Tripathy R, Fitzpatrick J, et al.
Biol Open
. 2023 Sep;
12(7).
PMID: 37746814
Intellectual disability is a neurodevelopmental disorder that affects 2-3% of the general population. Syndromic forms of intellectual disability frequently have a genetic basis and are often accompanied by additional developmental...
7.
Amrute J, Lai L, Ma P, Koenig A, Kamimoto K, Bredemeyer A, et al.
Nat Cardiovasc Res
. 2023 Aug;
2(4):399-416.
PMID: 37583573
Recovery of cardiac function is the holy grail of heart failure therapy yet is infrequently observed and remains poorly understood. In this study, we performed single-nucleus RNA sequencing from patients...
8.
Amrute J, Luo X, Penna V, Bredemeyer A, Yamawaki T, Yang S, et al.
Res Sq
. 2023 Feb;
PMID: 36747878
Inflammation and tissue fibrosis co-exist and are causally linked to organ dysfunction. However, the molecular mechanisms driving immune-fibroblast crosstalk in human cardiac disease remains unexplored and there are currently no...
9.
Koenig A, Shchukina I, Amrute J, Andhey P, Zaitsev K, Lai L, et al.
Nat Cardiovasc Res
. 2022 Aug;
1(3):263-280.
PMID: 35959412
Heart failure represents a major cause of morbidity and mortality worldwide. Single-cell transcriptomics have revolutionized our understanding of cell composition and associated gene expression. Through integrated analysis of single-cell and...
10.
Kopecky B, Dun H, Amrute J, Lin C, Bredemeyer A, Terada Y, et al.
Circulation
. 2022 Jul;
146(8):623-638.
PMID: 35880523
Background: Cellular rejection after heart transplantation imparts significant morbidity and mortality. Current immunosuppressive strategies are imperfect, target recipient T cells, and have adverse effects. The innate immune response plays an...