Amal K Dutta
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Explore the profile of Amal K Dutta including associated specialties, affiliations and a list of published articles.
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15
Citations
354
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Recent Articles
1.
Angsutararux P, Dutta A, Marras M, Abella C, Mellor R, Shi J, et al.
J Gen Physiol
. 2023 Mar;
155(5).
PMID: 36944081
Voltage-gated sodium (NaV) channels are responsible for the initiation and propagation of action potentials. In the heart, the predominant NaV1.5 α subunit is composed of four homologous repeats (I-IV) and...
2.
Dutta A, Boggs K, Khimji A, Getachew Y, Wang Y, Kresge C, et al.
Am J Physiol Gastrointest Liver Physiol
. 2020 Feb;
318(4):G763-G771.
PMID: 32090602
TMEM16A is a Ca-activated Cl channel in the apical membrane of biliary epithelial cells, known as cholangiocytes, which contributes importantly to ductular bile formation. Whereas cholangiocyte TMEM16A activity is regulated...
3.
Dutta A, Khimji A, Liu S, Karamysheva Z, Fujita A, Kresge C, et al.
Am J Physiol Gastrointest Liver Physiol
. 2015 Nov;
310(1):G34-42.
PMID: 26542395
TMEM16A is a newly identified Ca(2+)-activated Cl(-) channel in biliary epithelial cells (BECs) that is important in biliary secretion. While extracellular ATP stimulates TMEM16A via binding P2 receptors and increasing...
4.
Dutta A, Woo K, Khimji A, Kresge C, Feranchak A
Am J Physiol Gastrointest Liver Physiol
. 2012 Oct;
304(1):G87-98.
PMID: 23104560
Bile formation by the liver is initiated by canalicular transport at the hepatocyte membrane, leading to an increase in ductular bile flow. Thus, bile duct epithelial cells (cholangiocytes), which contribute...
5.
Dutta A, Khimji A, Kresge C, Bugde A, Dougherty M, Esser V, et al.
J Biol Chem
. 2010 Nov;
286(1):766-76.
PMID: 21041307
Cl(-) channels in the apical membrane of biliary epithelial cells (BECs) provide the driving force for ductular bile formation. Although a cystic fibrosis transmembrane conductance regulator has been identified in...
6.
Sarkar S, Greenleaf J, Gupta A, Ghosh D, Blaney L, Bandyopadhyay P, et al.
Water Res
. 2010 Aug;
44(19):5813-22.
PMID: 20728196
In Bangladesh and the neighboring state of West Bengal, India, over 100 million people are affected by widespread arsenic poisoning through drinking water drawn from underground sources containing arsenic at...
7.
Dutta A, Khimji A, Sathe M, Kresge C, Parameswara V, Esser V, et al.
Am J Physiol Gastrointest Liver Physiol
. 2010 May;
297(5):G1009-18.
PMID: 20501432
In the liver, adenosine triphosphate (ATP) is an extracellular signaling molecule that is released into bile and stimulates a biliary epithelial cell secretory response via engagement of apical P2 receptors....
8.
Simpson L, Burdine L, Dutta A, Feranchak A, Kodadek T
J Am Chem Soc
. 2009 Apr;
131(16):5760-2.
PMID: 19351156
Bacterial toxin-mediated diarrheal disease is a major cause of morbidity and mortality worldwide. In this work we designed an on-bead library of protease-resistant, acid-stable peptoid molecules and screened for high...
9.
Dutta A, Woo K, Brian Doctor R, Fitz J, Feranchak A
Am J Physiol Gastrointest Liver Physiol
. 2008 Sep;
295(5):G1004-15.
PMID: 18787062
Extracellular ATP regulates bile formation by binding to P2 receptors on cholangiocytes and stimulating transepithelial Cl(-) secretion. However, the specific signaling pathways linking receptor binding to Cl(-) channel activation are...
10.
Woo K, Dutta A, Patel V, Kresge C, Feranchak A
J Physiol
. 2008 Apr;
586(11):2779-98.
PMID: 18388137
ATP in bile is a potent secretogogue, stimulating cholangiocyte Cl- and fluid secretion via binding to membrane P2 receptors, though the physiological stimuli involved in biliary ATP release are unknown....