Yuma Horii
Overview
Explore the profile of Yuma Horii including associated specialties, affiliations and a list of published articles.
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Articles
9
Citations
133
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Recent Articles
1.
Horii Y, Matsuda S, Toyota C, Morinaga T, Nakaya T, Tsuchiya S, et al.
Nat Commun
. 2023 Feb;
14(1):550.
PMID: 36754961
Myofibroblasts cause tissue fibrosis by producing extracellular matrix proteins, such as collagens. Humoral factors like TGF-β, and matrix stiffness are important for collagen production by myofibroblasts. However, the molecular mechanisms...
2.
Hironaka T, Takizawa N, Yamauchi Y, Horii Y, Nakaya M
J Biol Chem
. 2023 Jan;
299(3):102934.
PMID: 36690273
Fibrosis is mainly triggered by inflammation in various tissues, such as heart and liver tissues, and eventually leads to their subsequent dysfunction. Fibrosis is characterized by the excessive accumulation of...
3.
Horii Y, Matsuda S, Watari K, Nagasaka A, Kurose H, Nakaya M
Bio Protoc
. 2021 Sep;
7(18):e2553.
PMID: 34541199
In myocardial infarction (MI), a number of cardiomyocytes undergo apoptosis. These apoptotic cardiomyocytes are promptly engulfed by phagocytes. If the dead cells are not engulfed, their noxious contents are released...
4.
Horii Y, Matsuda S, Watari K, Nagasaka A, Kurose H, Nakaya M
Bio Protoc
. 2021 Sep;
7(18):e2552.
PMID: 34541198
In myocardial infarction (MI), a plenty of cardiomyocytes undergo necrosis and necroptosis due to the lack of oxygen and nutrients. The dead cardiomyocytes are promptly engulfed by phagocytes. When the...
5.
Takizawa N, Hironaka T, Mae K, Ueno T, Horii Y, Nagasaka A, et al.
Biochem Biophys Res Commun
. 2021 May;
561:180-186.
PMID: 34023784
Fibrosis is a condition characterized by the overproduction of extracellular matrix (ECM) components (e.g., collagen) in the myofibroblasts, causing tissue hardening and eventual organ dysfunction. Currently, the molecular mechanisms that...
6.
Hironaka T, Ueno T, Mae K, Yoshimura C, Morinaga T, Horii Y, et al.
Biochem Biophys Res Commun
. 2020 Jul;
529(2):224-230.
PMID: 32703415
Fibrosis is attributed to excess deposition of extracellular matrix (ECM) proteins including collagen and is associated with various organ dysfunction. This excessive ECM is produced by myofibroblasts, which are differentiated...
7.
Horii Y, Nakaya M, Ohara H, Nishihara H, Watari K, Nagasaka A, et al.
FASEB J
. 2020 May;
34(6):8749-8763.
PMID: 32385915
Leukotriene B receptor 1 (BLT1), a high-affinity G-protein-coupled receptor for leukotriene B4 (LTB ), is expressed on various inflammatory cells and plays critical roles in several inflammatory diseases. In myocardial...
8.
Nagasaka A, Mogi C, Ono H, Nishi T, Horii Y, Ohba Y, et al.
Sci Rep
. 2017 Aug;
7(1):7812.
PMID: 28798316
Myocardial infarction (MI) is an ischaemic heart condition caused by the occlusion of coronary arteries. Following MI, lactic acid from anaerobic glycolysis increases and infiltrating immune cells produce severe inflammation,...
9.
Nakaya M, Watari K, Tajima M, Nakaya T, Matsuda S, Ohara H, et al.
J Clin Invest
. 2016 Dec;
127(1):383-401.
PMID: 27918308
Myocardial infarction (MI) results in the generation of dead cells in the infarcted area. These cells are swiftly removed by phagocytes to minimize inflammation and limit expansion of the damaged...