Yosei Miki
Overview
Explore the profile of Yosei Miki including associated specialties, affiliations and a list of published articles.
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Articles
6
Citations
13
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0
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Recent Articles
1.
Sato Y, Habara M, Hanaki S, Masaki T, Tomiyasu H, Miki Y, et al.
Proc Natl Acad Sci U S A
. 2024 Oct;
121(41):e2414618121.
PMID: 39361641
The transcription factor E2F1 serves as a regulator of the cell cycle and promotes cell proliferation. It is highly expressed in cancer tissues and contributes to their malignant transformation. Degradation...
2.
Sato Y, Habara M, Hanaki S, Sharif J, Tomiyasu H, Miki Y, et al.
Sci Rep
. 2024 Jun;
14(1):14732.
PMID: 38926604
Excess amounts of histones in the cell induce mitotic chromosome loss and genomic instability, and are therefore detrimental to cell survival. In yeast, excess histones are degraded by the proteasome...
3.
Hanaki S, Habara M, Tomiyasu H, Sato Y, Miki Y, Masaki T, et al.
Life Sci Alliance
. 2024 May;
7(8).
PMID: 38803221
FK506-binding protein 52 (FKBP52) is a member of the FKBP family of proline isomerases. FKBP52 is up-regulated in various cancers and functions as a positive regulator of steroid hormone receptors....
4.
Tomiyasu H, Habara M, Hanaki S, Sato Y, Miki Y, Shimada M
J Biol Chem
. 2024 Mar;
300(4):107209.
PMID: 38519029
FOXO1 is a transcription factor and potential tumor suppressor that is negatively regulated downstream of PI3K-PKB/AKT signaling. Paradoxically, FOXO also promotes tumor growth, but the detailed mechanisms behind this role...
5.
Hanaki S, Habara M, Sato Y, Tomiyasu H, Miki Y, Shibutani S, et al.
J Biochem
. 2023 Nov;
175(3):235-244.
PMID: 38030387
The transcription factor NFAT plays key roles in multiple biological activities, such as immune responses, tissue development and malignant transformation. NFAT is dephosphorylated by calcineurin, which is activated by intracellular...
6.
Masaki T, Habara M, Hanaki S, Sato Y, Tomiyasu H, Miki Y, et al.
Sci Rep
. 2023 Aug;
13(1):13116.
PMID: 37573463
c-Myc, a transcription factor, induces cell proliferation and is often aberrantly or highly expressed in cancers. However, molecular mechanisms underlying this aberrantly high expression remain unclear. Here, we found that...