Y Shiloh
Overview
Explore the profile of Y Shiloh including associated specialties, affiliations and a list of published articles.
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Articles
139
Citations
6038
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Recent Articles
11.
Gatei M, Shkedy D, Khanna K, Uziel T, Shiloh Y, Pandita T, et al.
Oncogene
. 2001 Apr;
20(3):289-94.
PMID: 11313957
Cells from patients with the genetic disorder ataxia-telangiectasia (A-T) are hypersensitive to ionizing radiation and radiomimetic agents, both of which generate reactive oxygen species capable of causing oxidative damage to...
12.
Peter Y, Rotman G, Lotem J, Elson A, Shiloh Y, Groner Y
EMBO J
. 2001 Apr;
20(7):1538-46.
PMID: 11285218
Patients with the genetic disorder ataxia-telangiectasia (A-T) display a pleiotropic phenotype that includes neurodegeneration, immunodeficiency, cancer predisposition and hypersensitivity to ionizing radiation. The gene responsible is ATM, and ATM:-knockout mice...
13.
Buschmann T, Potapova O, Bar-Shira A, Ivanov V, Fuchs S, Henderson S, et al.
Mol Cell Biol
. 2001 Apr;
21(8):2743-54.
PMID: 11283254
The p53 tumor suppressor protein plays a key role in the regulation of stress-mediated growth arrest and apoptosis. Stress-induced phosphorylation of p53 tightly regulates its stability and transcriptional activities. Mass...
14.
Kamsler A, Daily D, HOCHMAN A, Stern N, Shiloh Y, Rotman G, et al.
Cancer Res
. 2001 Mar;
61(5):1849-54.
PMID: 11280737
Ataxia-telangiectasia (A-T) is a genetic disorder caused by mutational inactivation of the ATM gene. A-T patients display a pleiotropic phenotype and suffer primarily from progressive ataxia caused by degeneration of...
15.
Shiloh Y
Curr Opin Genet Dev
. 2001 Feb;
11(1):71-7.
PMID: 11163154
Maintenance of genome stability depends on the appropriate response to DNA damage. This response is based on complex networks of signaling pathways that activate numerous processes and lead ultimately to...
16.
Li N, Banin S, Ouyang H, Li G, Courtois G, Shiloh Y, et al.
J Biol Chem
. 2000 Dec;
276(12):8898-903.
PMID: 11114307
Following challenge with proinflammatory stimuli or generation of DNA double strand breaks (DSBs), transcription factor NF-kappaB translocates from the cytoplasm to the nucleus to activate expression of target genes. In...
17.
Matsuoka S, Rotman G, Ogawa A, Shiloh Y, Tamai K, Elledge S
Proc Natl Acad Sci U S A
. 2000 Sep;
97(19):10389-94.
PMID: 10973490
The protein kinase Chk2, the mammalian homolog of the budding yeast Rad53 and fission yeast Cds1 checkpoint kinases, is phosphorylated and activated in response to DNA damage by ionizing radiation...
18.
Li S, TING N, Zheng L, Chen P, Ziv Y, Shiloh Y, et al.
Nature
. 2000 Jul;
406(6792):210-5.
PMID: 10910365
BRCA1 encodes a familial breast cancer suppressor that has a critical role in cellular responses to DNA damage. Mouse cells deficient for Brca1 show genetic instability, defective G2-M checkpoint control...
19.
Ziv Y, Banin S, Lim D, Canman C, Kastan M, Shiloh Y
Methods Mol Biol
. 2000 Jul;
99:99-108.
PMID: 10909080
No abstract available.
20.
Zhao S, Weng Y, Yuan S, Lin Y, Hsu H, Lin S, et al.
Nature
. 2000 Jun;
405(6785):473-7.
PMID: 10839544
Ataxia-telangiectasia (A-T) and Nijmegen breakage syndrome (NBS) are recessive genetic disorders with susceptibility to cancer and similar cellular phenotypes. The protein product of the gene responsible for A-T, designated ATM,...