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Xu Shi-Wen

Explore the profile of Xu Shi-Wen including associated specialties, affiliations and a list of published articles. Areas
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Articles 52
Citations 2065
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Recent Articles
1.
Quesnel K, Shi-Wen X, Hutchenreuther J, Xiao Y, Liu S, Peidl A, et al.
Matrix Biol Plus . 2021 Feb; 3:100009. PMID: 33543008
The microenvironment contributes to the excessive connective tissue deposition that characterizes fibrosis. Members of the CCN family of matricellular proteins are secreted by fibroblasts into the fibrotic microenvironment; however, the...
2.
Shi-Wen X, Racanelli M, Ali A, Simon A, Quesnel K, Stratton R, et al.
J Cell Commun Signal . 2021 Jan; 15(1):71-80. PMID: 33398723
Fibrosis is perpetuated by an autocrine, pro-adhesive signaling loop maintained by the synthetic and contractile abilities of myofibroblasts and the stiff, highly-crosslinked extracellular matrix. Transcriptional complexes that are exquisitely responsive...
3.
Murphy-Marshman H, Quensel K, Shi-Wen X, Barnfield R, Kelly J, Peidl A, et al.
PLoS One . 2017 Oct; 12(10):e0186740. PMID: 29049376
TGFbeta induces fibrogenic responses in fibroblasts. Reactive oxygen species (ROS)/nicotinamide adenine dinucleotide phosphate (NADPH) oxidase (NOX) may contribute to fibrogenic responses. Here, we examine if the antioxidant N-acetylcysteine (NAC), the...
4.
Ikeda T, Fragiadaki M, Shi-Wen X, Ponticos M, Khan K, Denton C, et al.
Biochem Biophys Rep . 2017 Sep; 7:246-252. PMID: 28955913
In the enhancer region of the human type I collagen alpha 2 () gene, we identified cis-elements for the transcription factor CUX1. However, the role of CUX1 in fibrosis remains...
5.
Ikeda T, Fragiadaki M, Shi-Wen X, Ponticos M, Khan K, Denton C, et al.
Data Brief . 2016 Sep; 8:1377-80. PMID: 27583344
This data article contains complementary figures related to the research article entitled, "Transforming growth factor-β-induced CUX1 isoforms are associated with fibrosis in systemic sclerosis lung fibroblasts" (Ikeda et al. (2016)...
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Lindahl G, Stock C, Shi-Wen X, Leoni P, Sestini P, Howat S, et al.
Respir Res . 2013 Aug; 14:80. PMID: 23915349
Background: Interstitial lung disease is a major cause of morbidity and mortality in systemic sclerosis (SSc), with insufficiently effective treatment options. Progression of pulmonary fibrosis involves expanding populations of fibroblasts,...
8.
Shi-Wen X, Thompson K, Khan K, Liu S, Murphy-Marshman H, Baron M, et al.
Rheumatology (Oxford) . 2012 Sep; 51(12):2146-54. PMID: 22977060
Objective: Fibrotic diseases such as SSc (systemic sclerosis, scleroderma) are characterized by the abnormal presence of the myofibroblast, a specialized type of fibroblast that overexpresses the highly contractile protein α-smooth...
9.
Thoua N, Derrett-Smith E, Khan K, Dooley A, Shi-Wen X, Denton C
Rheumatology (Oxford) . 2012 Aug; 51(11):1989-98. PMID: 22908328
Objective: Gastrointestinal involvement occurs in up to 90% of patients with SSc. Animal models of SSc mimic some of the pathophysiological disease processes of SSc. The transgenic (TG) mouse strain...
10.
Dreifaldt M, Souza D, Bodin L, Shi-Wen X, Dooley A, Muddle J, et al.
Angiology . 2012 May; 64(4):293-9. PMID: 22569406
No-touch (NT) saphenous vein (SV) grafts are superior to SVs harvested by the conventional technique (CT), with a patency comparable with the internal thoracic artery (ITA). Preservation of the vasa...