Vincent H Gattone
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Explore the profile of Vincent H Gattone including associated specialties, affiliations and a list of published articles.
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15
Citations
481
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Recent Articles
1.
Parchem J, Kanasaki K, Kanasaki M, Sugimoto H, Xie L, Hamano Y, et al.
J Clin Invest
. 2018 Sep;
128(11):5008-5017.
PMID: 30179860
Preeclampsia remains a clinical challenge due to its poorly understood pathogenesis. A prevailing notion is that increased placental production of soluble fms-like tyrosine kinase-1 (sFlt-1) causes the maternal syndrome by...
2.
Flaig S, Gattone V, Blazer-Yost B
J Transl Int Med
. 2017 Feb;
4(3):118-126.
PMID: 28191533
Background And Objectives: The studies were designed to test the efficacy of two peroxisome proliferator-activated receptor γ (PPARγ) agonists in two rodent models of polycystic kidney disease (PKD). Materials And...
3.
Jarial M, Gattone V, Wilkins J
Zoolog Sci
. 2014 May;
31(5):283-91.
PMID: 24832901
The morphology of the nephrons of the coelacanth Latimeria chalumnae was investigated by electron microscopy. Each nephron is composed of a large renal corpuscle with well vascularized glomerulus, ciliated neck...
4.
Werner M, Ward H, Phillips C, Miller C, Gattone V, Bacallao R
Am J Physiol Cell Physiol
. 2013 Mar;
305(1):C36-47.
PMID: 23515530
Mutations in inversin cause nephronophthisis type II, an autosomal recessive form of polycystic kidney disease associated with situs inversus, dilatation, and kidney cyst formation. Since cyst formation may represent a...
5.
Chen N, ONeill K, Chen X, Kiattisunthorn K, Gattone V, Moe S
Am J Nephrol
. 2013 Mar;
37(3):191-8.
PMID: 23466870
Background: Transglutaminase 2 (TGM2) is a calcium-dependent enzyme that can cross-link nearly all extracellular matrix (ECM) proteins and can facilitate cell-ECM interaction through integrins. Given the importance of the ECM...
6.
Blazer-Yost B, Blacklock B, Flaig S, Bacallao R, Gattone V
Cell Physiol Biochem
. 2011 Dec;
28(6):1255-64.
PMID: 22179013
Autosomal dominant polycystic kidney disease (ADPKD) is characterized by the slow growth of multiple fluid-filled cysts predominately in the kidney tubules and liver bile ducts. Elucidation of mechanisms that control...
7.
Chen N, ONeill K, Chen X, Kiattisunthorn K, Gattone V, Moe S
Am J Nephrol
. 2011 Jul;
34(3):211-9.
PMID: 21791917
Background: The objective of the current study was to determine if altered regulation of matrix metalloproteinases (MMPs) may predispose to extracellular matrix degradation, facilitating arterial calcification in chronic kidney disease...
8.
Mason S, Lai X, Bacallao R, Blazer-Yost B, Gattone V, Wang K, et al.
Proteomics Clin Appl
. 2010 Jun;
3(10):1247-1250.
PMID: 20526430
Autosomal Dominant Polycystic Kidney Disease (ADPKD) is characterized by the development of numerous fluid-filled cysts in the kidneys of patients. We recently published our description of the proteome of renal...
9.
Kanasaki K, Palmsten K, Sugimoto H, Ahmad S, Hamano Y, Xie L, et al.
Nature
. 2008 May;
453(7198):1117-21.
PMID: 18469803
Despite intense investigation, mechanisms that facilitate the emergence of the pre-eclampsia phenotype in women are still unknown. Placental hypoxia, hypertension, proteinuria and oedema are the principal clinical features of this...
10.
Amura C, Brodsky K, Groff R, Gattone V, Voelkel N, Brian Doctor R
Am J Physiol Cell Physiol
. 2007 May;
293(1):C419-28.
PMID: 17475663
Proliferation of cyst-lining epithelial cells is an integral part of autosomal dominant polycystic kidney disease (ADPKD) cyst growth. Cytokines and growth factors within cyst fluids are positioned to induce cyst...