Vanessa Rodrik
Overview
Explore the profile of Vanessa Rodrik including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
6
Citations
234
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
OReilly K, Warycha M, Davies M, Rodrik V, Zhou X, Yee H, et al.
Clin Cancer Res
. 2009 Apr;
15(8):2872-8.
PMID: 19336517
Purpose: Both phosphatidylinositol 3-kinase/AKT and RAS/mitogen-activated protein kinase signal transduction pathways mediate 4E-BP1 phosphorylation, releasing 4E-BP1 from the mRNA cap and permitting translation initiation. Given the prevalence of PTEN and...
2.
Rodrik V, Gomes E, Hui L, Rockwell P, Foster D
FEBS Lett
. 2006 Sep;
580(24):5647-52.
PMID: 16996503
Estrogen, which has been strongly implicated in breast cancer, suppresses apoptosis in estrogen receptor (ER) positive MCF-7 breast cancer cells. Phospholipase D (PLD), which is commonly elevated in ER negative...
3.
Zheng Y, Rodrik V, Toschi A, Shi M, Hui L, Shen Y, et al.
J Biol Chem
. 2006 Apr;
281(23):15862-8.
PMID: 16595654
MDA-MB-231 human breast cancer cells belong to a highly invasive metastatic cell line that depends on phospholipase D (PLD) activity for survival when deprived of serum growth factors. In response...
4.
Hui L, Rodrik V, Pielak R, Knirr S, Zheng Y, Foster D
J Biol Chem
. 2005 Aug;
280(43):35829-35.
PMID: 16109716
A critical aspect of tumor progression is the generation of survival signals that overcome default apoptotic programs. Recent studies have revealed that elevated phospholipase D activity generates survival signals in...
5.
Rodrik V, Zheng Y, Harrow F, Chen Y, Foster D
Mol Cell Biol
. 2005 Aug;
25(17):7917-25.
PMID: 16107734
Estrogens, which have been strongly implicated in the development of breast cancer, enhance proliferation of mammary epithelial cells and, importantly, estrogen receptor (ER)-positive breast cancer cells. In the absence of...
6.
Chen Y, Rodrik V, Foster D
Oncogene
. 2004 Dec;
24(4):672-9.
PMID: 15580312
Cancer cells generate survival signals to suppress default apoptotic programs that protect from cancer. Phosphatidylinositol-3-kinase (PI3K) generates a survival signal that is frequently dysregulated in human cancers. Phospholipase D (PLD)...