Suvendra N Bhattacharyya
Overview
Explore the profile of Suvendra N Bhattacharyya including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
35
Citations
5699
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Ray S, Roychowdhury S, Chakrabarty Y, Banerjee S, Hobbs A, Chattopadhyay K, et al.
Structure
. 2025 Mar;
PMID: 40056914
Phase separation into membrane-less organelles regulates protein activity in eukaryotic cells. miRNA-repressed mRNAs and Ago proteins localize to RNA-processing bodies (P-bodies), which are subcellular structures formed by several RNA-binding and...
2.
Hom Choudhury S, Bhattacharjee S, Mukherjee K, Bhattacharyya S
J Biol Chem
. 2024 Mar;
300(4):107170.
PMID: 38492777
Intercellular miRNA exchange acts as a key mechanism to control gene expression post-transcriptionally in mammalian cells. Regulated export of repressive miRNAs allows the expression of inflammatory cytokines in activated macrophages....
3.
Ghosh S, Hom Choudhury S, Mukherjee K, Bhattacharyya S
J Biol Chem
. 2024 Feb;
300(3):105750.
PMID: 38360271
Extracellular vesicles-mediated exchange of miRNA cargos between diverse types of mammalian cells is a major mechanism of controlling cellular miRNA levels and activity, thus regulating the expression of miRNA-target genes...
4.
Bandyopadhyay D, Basu S, Mukherjee I, Chakrabarti S, Chakrabarti P, Mukherjee K, et al.
J Biol Chem
. 2023 Jul;
299(8):104999.
PMID: 37394005
Hepatocytes on exposure to high levels of lipids reorganize the metabolic program while fighting against the toxicity associated with elevated cellular lipids. The mechanism of this metabolic reorientation and stress...
5.
6.
Chatterjee S, Mukherjee I, Bhattacharjee S, Bose M, Chakrabarti S, Bhattacharyya S
Mol Cell Biol
. 2022 Mar;
42(4):e0045221.
PMID: 35311564
MicroRNAs (miRNAs) repress protein expression by binding to the target mRNAs. Exploring whether the expression of one miRNA can regulate the abundance and activity of other miRNAs, we noted the...
7.
Ganguly S, Ghoshal B, Banerji I, Bhattacharjee S, Chakraborty S, Goswami A, et al.
Life Sci Alliance
. 2022 Feb;
5(6).
PMID: 35210329
, the causative agent of visceral leishmaniasis, infects and resides within tissue macrophage cells. It is not clear how the parasite infected cells crosstalk with the noninfected cells to regulate...
8.
DAS A, Basu S, Bandyopadhyay D, Mukherjee K, Datta D, Chakraborty S, et al.
iScience
. 2021 Dec;
24(12):103428.
PMID: 34877493
Hepatic miRNA, miR-122, plays an important role in controlling metabolic homeostasis in mammalian liver. Intercellular transfer of miR-122 was found to play a role in controlling tissue inflammation. miR-122, as...
9.
De D, Bhattacharyya S
J Cell Sci
. 2021 Jun;
134(11).
PMID: 34096603
Upon exposure to amyloid-β oligomers (Aβ1-42), glial cells start expressing proinflammatory cytokines, despite an increase in levels of repressive microRNAs (miRNAs). Exploring the mechanism of this potential immunity of target...
10.
De D, Mukherjee I, Guha S, Paidi R, Chakrabarti S, Biswas S, et al.
Mol Ther Nucleic Acids
. 2021 Jun;
24:868-887.
PMID: 34094708
Deposition of amyloid beta plaques in adult rat or human brain is associated with increased production of proinflammatory cytokines by associated glial cells that are responsible for degeneration of the...