Susanne Homann
Overview
Explore the profile of Susanne Homann including associated specialties, affiliations and a list of published articles.
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10
Citations
475
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Recent Articles
1.
Hartmann F, Gorski D, Newman A, Homann S, Petz A, Owsiany K, et al.
Circ Res
. 2021 Oct;
129(11):992-1005.
PMID: 34615369
[Figure: see text].
2.
Vogel J, Boehme P, Homann S, Boehm M, Schutt K, Boden K, et al.
Hypertens Res
. 2021 Sep;
44(12):1568-1577.
PMID: 34548653
Therapy-resistant hypertension is a serious medical problem, causing end-organ damage, stroke, and heart failure if untreated. Since the standard of care fails in resistant hypertension patients, there is still a...
3.
Hackert K, Homann S, Mir S, Beran A, Gorressen S, Funk F, et al.
Hypertension
. 2021 Mar;
77(6):1918-1927.
PMID: 33745300
[Figure: see text].
4.
Tomasovic A, Brand T, Schanbacher C, Kramer S, Hummert M, Godoy P, et al.
Nat Commun
. 2020 Apr;
11(1):1733.
PMID: 32265441
Dysregulation of extracellular signal-regulated kinases (ERK1/2) is linked to several diseases including heart failure, genetic syndromes and cancer. Inhibition of ERK1/2, however, can cause severe cardiac side-effects, precluding its wide...
5.
Petz A, Grandoch M, Gorski D, Abrams M, Piroth M, Schneckmann R, et al.
Circ Res
. 2019 Mar;
124(10):1433-1447.
PMID: 30916618
Rationale: Immediate changes in the ECM (extracellular matrix) microenvironment occur after myocardial ischemia and reperfusion (I/R) injury. Objective: Aim of this study was to unravel the role of the early...
6.
Bartolomaeus H, Balogh A, Yakoub M, Homann S, Marko L, Hoges S, et al.
Circulation
. 2018 Dec;
139(11):1407-1421.
PMID: 30586752
Background: Arterial hypertension and its organ sequelae show characteristics of T cell-mediated inflammatory diseases. Experimental anti-inflammatory therapies have been shown to ameliorate hypertensive end-organ damage. Recently, the CANTOS study (Canakinumab...
7.
Homann S, Grandoch M, Kiene L, Podsvyadek Y, Feldmann K, Rabausch B, et al.
Matrix Biol
. 2017 Oct;
66:67-80.
PMID: 28987865
Objective: Hyaluronan (HA) is a prominent component of the provisional extracellular matrix (ECM) present in the neointima of atherosclerotic plaques. Here the role of HA synthase 3 (HAS3) in atheroprogression...
8.
Grandoch M, Kohlmorgen C, Melchior-Becker A, Feldmann K, Homann S, Muller J, et al.
Arterioscler Thromb Vasc Biol
. 2016 Apr;
36(5):e41-50.
PMID: 27034473
Objective: Thrombin signaling promotes atherosclerosis by initiating inflammatory events indirectly through platelet activation and directly via protease-activated receptors. Therefore, endogenous thrombin inhibitors may be relevant modulators of atheroprogression and cardiovascular...
9.
Kiene L, Homann S, Suvorava T, Rabausch B, Muller J, Kojda G, et al.
Arterioscler Thromb Vasc Biol
. 2015 Nov;
36(2):e9-16.
PMID: 26586662
Objective: Hyaluronan (HA) is a polymeric glucosaminoglycan that forms a provisional extracellular matrix in diseased vessels. HA is synthesized by 3 different HA synthases (HAS1, HAS2, and HAS3). Aim of...
10.
Grandoch M, Feldmann K, Gothert J, Dick L, Homann S, Klatt C, et al.
Circ Res
. 2015 Mar;
116(8):e57-68.
PMID: 25740843
Rationale: Lymphotoxin β receptor (LTbR) regulates immune cell trafficking and communication in inflammatory diseases. However, the role of LTbR in atherosclerosis is still unclear. Objective: The aim of this study...