Stephen Crimmins
Overview
Explore the profile of Stephen Crimmins including associated specialties, affiliations and a list of published articles.
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Articles
5
Citations
289
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0
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Recent Articles
1.
Crimmins S, Smiley R, Preston K, Yau A, McCallum R, Showkat Ali M
Gastroenterology Res
. 2016 Oct;
9(2-3):39-46.
PMID: 27785323
Background: Gastroparesis is a significant co-morbidity affecting up to 50% of patients with diabetes and is disproportionately found in women. Prior studies have suggested that loss of interstitial cells of...
2.
Qiao L, Hamamichi S, Caldwell K, Caldwell G, Yacoubian T, Wilson S, et al.
Mol Brain
. 2008 Nov;
1:17.
PMID: 19021916
α-synuclein (α-syn) is a main component of Lewy bodies (LB) that occur in many neurodegenerative diseases, including Parkinson's disease (PD), dementia with LB (DLB) and multi-system atrophy. α-syn mutations or...
3.
Crimmins S, Sutovsky M, Chen P, Huffman A, Wheeler C, Swing D, et al.
Dev Biol
. 2008 Oct;
325(1):33-42.
PMID: 18926813
Homozygous ataxia (ax(J)) mice have reduced expression of ubiquitin-specific protease 14 (Usp14), resulting in severe neuromuscular defects and death by 2 months of age. Transgenic expression of Usp14 exclusively in...
4.
Transgenic rescue of ataxia mice with neuronal-specific expression of ubiquitin-specific protease 14
Crimmins S, Jin Y, Wheeler C, Huffman A, Chapman C, Dobrunz L, et al.
J Neurosci
. 2006 Nov;
26(44):11423-31.
PMID: 17079671
The ataxia mutation (axJ) is a recessive neurological mutation that results in reduced growth, ataxia, and hindlimb muscle wasting in mice. The axJ gene encodes ubiquitin-specific protease 14 (Usp14), a...
5.
Anderson C, Crimmins S, Wilson J, Korbel G, Ploegh H, Wilson S
J Neurochem
. 2005 Sep;
95(3):724-31.
PMID: 16190881
The ataxia (ax(J)) mutation is a spontaneous recessive mutation that results in reduced expression of ubiquitin-specific protease 14, Usp14. Mice homozygous for the ax(J) mutation are retarded for growth and...