Souradipta Ganguly
Overview
Explore the profile of Souradipta Ganguly including associated specialties, affiliations and a list of published articles.
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11
Citations
153
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Recent Articles
1.
Gu L, Zhu Y, Nandi S, Lee M, Watari K, Bareng B, et al.
Nature
. 2025 Jan;
638(8051):E30.
PMID: 39890899
No abstract available.
2.
Varanasi S, Chen D, Liu Y, Johnson M, Miller C, Ganguly S, et al.
Science
. 2025 Jan;
387(6730):192-201.
PMID: 39787217
The metabolic landscape of cancer greatly influences antitumor immunity, yet it remains unclear how organ-specific metabolites in the tumor microenvironment influence immunosurveillance. We found that accumulation of primary conjugated and...
3.
Cong M, Carvalho Gontijo Weber R, Sakane S, Zhang V, Jiang C, Taura K, et al.
Hepatol Commun
. 2025 Jan;
8(11).
PMID: 39761005
Background: Liver fibrosis is caused by chronic toxic or cholestatic liver injury. Fibrosis results from the recruitment of myeloid cells into the injured liver, the release of inflammatory and fibrogenic...
4.
Gu L, Zhu Y, Nandi S, Lee M, Watari K, Bareng B, et al.
Nature
. 2025 Jan;
637(8045):461-469.
PMID: 39743585
Hepatocellular carcinoma (HCC) originates from differentiated hepatocytes undergoing compensatory proliferation in livers damaged by viruses or metabolic-dysfunction-associated steatohepatitis (MASH). While increasing HCC risk, MASH triggers p53-dependent hepatocyte senescence, which we...
5.
Lipid-associated macrophages' promotion of fibrosis resolution during MASH regression requires TREM2
Ganguly S, Rosenthal S, Ishizuka K, Troutman T, Rohm T, Khader N, et al.
Proc Natl Acad Sci U S A
. 2024 Aug;
121(35):e2405746121.
PMID: 39172787
While macrophage heterogeneity during metabolic dysfunction-associated steatohepatitis (MASH) has been described, the fate of these macrophages during MASH regression is poorly understood. Comparing macrophage heterogeneity during MASH progression vs regression,...
6.
Osna N, Tikhanovich I, Ortega-Ribera M, Mueller S, Zheng C, Mueller J, et al.
Biomolecules
. 2024 Apr;
14(4.
PMID: 38672422
Alcohol-associated liver disease (ALD) is a substantial cause of morbidity and mortality worldwide and represents a spectrum of liver injury beginning with hepatic steatosis (fatty liver) progressing to inflammation and...
7.
Ganguly S, Muench G, Shang L, Rosenthal S, Rahman G, Wang R, et al.
Cell Mol Gastroenterol Hepatol
. 2021 Jun;
12(3):891-920.
PMID: 34062281
Background & Aims: How benign liver steatosis progresses to nonalcoholic steatohepatitis (NASH), fibrosis, and hepatocellular carcinoma (HCC) remains elusive. NASH progression entails diverse pathogenic mechanisms and relies on complex cross-talk...
8.
Rosenthal S, Liu X, Ganguly S, Dhar D, Pasillas M, Ricciardelli E, et al.
Hepatology
. 2021 Feb;
74(2):667-685.
PMID: 33550587
Background And Aims: In clinical and experimental NASH, the origin of the scar-forming myofibroblast is the HSC. We used foz/foz mice on a Western diet to characterize in detail the...
9.
Western Diet Promotes Renal Injury, Inflammation, and Fibrosis in a Murine Model of Alström Syndrome
Kim Y, Ganguly S, Nespoux J, Freeman B, Zhang H, Brenner D, et al.
Nephron
. 2020 Jul;
144(8):400-412.
PMID: 32629454
Introduction: Alström syndrome is a rare recessive genetic disease caused by mutations in ALMS1, which encodes a protein that is related to cilia function and intracellular endosome trafficking. The syndrome...
10.
Liang S, Ma H, Zhong Z, Dhar D, Liu X, Xu J, et al.
Gastroenterology
. 2018 Nov;
156(4):1156-1172.e6.
PMID: 30445007
Background & Aims: Although there are associations among oxidative stress, reduced nicotinamide adenine dinucleotide phosphate oxidase (NOX) activation, and hepatocellular carcinoma (HCC) development, it is not clear how NOX contributes...