Marcos G Teneche
Overview
Explore the profile of Marcos G Teneche including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
6
Citations
19
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Miller K, Li B, Pierce-Hoffman H, Patel S, Lei X, Rajesh A, et al.
Nat Commun
. 2025 Mar;
16(1):2229.
PMID: 40044657
Genomic instability and inflammation are distinct hallmarks of aging, but the connection between them is poorly understood. Here we report a mechanism directly linking genomic instability and inflammation in senescent...
2.
Gu L, Zhu Y, Nandi S, Lee M, Watari K, Bareng B, et al.
Nature
. 2025 Jan;
638(8051):E30.
PMID: 39890899
No abstract available.
3.
Varanasi S, Chen D, Liu Y, Johnson M, Miller C, Ganguly S, et al.
Science
. 2025 Jan;
387(6730):192-201.
PMID: 39787217
The metabolic landscape of cancer greatly influences antitumor immunity, yet it remains unclear how organ-specific metabolites in the tumor microenvironment influence immunosurveillance. We found that accumulation of primary conjugated and...
4.
Gu L, Zhu Y, Nandi S, Lee M, Watari K, Bareng B, et al.
Nature
. 2025 Jan;
637(8045):461-469.
PMID: 39743585
Hepatocellular carcinoma (HCC) originates from differentiated hepatocytes undergoing compensatory proliferation in livers damaged by viruses or metabolic-dysfunction-associated steatohepatitis (MASH). While increasing HCC risk, MASH triggers p53-dependent hepatocyte senescence, which we...
5.
Dasgupta N, Lei X, Shi C, Arnold R, Teneche M, Miller K, et al.
Mol Cell
. 2024 Aug;
84(17):3271-3287.e8.
PMID: 39178863
Cellular senescence, a stress-induced stable proliferation arrest associated with an inflammatory senescence-associated secretory phenotype (SASP), is a cause of aging. In senescent cells, cytoplasmic chromatin fragments (CCFs) activate SASP via...
6.
Dasgupta N, Lei X, Shi C, Arnold R, Teneche M, Miller K, et al.
bioRxiv
. 2024 Jul;
PMID: 38979156
Cellular senescence, a stress-induced stable proliferation arrest associated with an inflammatory Senescence-Associated Secretory Phenotype (SASP), is a cause of aging. In senescent cells, Cytoplasmic Chromatin Fragments (CCFs) activate SASP via...
7.
Suryadevara V, Hudgins A, Rajesh A, Pappalardo A, Karpova A, Dey A, et al.
Nat Rev Mol Cell Biol
. 2024 Jun;
25(12):1001-1023.
PMID: 38831121
Once considered a tissue culture-specific phenomenon, cellular senescence has now been linked to various biological processes with both beneficial and detrimental roles in humans, rodents and other species. Much of...
8.
Miller K, Li B, Pierce-Hoffman H, Patel S, Lei X, Rajesh A, et al.
bioRxiv
. 2023 Dec;
PMID: 38045344
Genomic instability and inflammation are distinct hallmarks of aging, but the connection between them is poorly understood. Understanding their interrelationship will help unravel new mechanisms and therapeutic targets of aging...
9.
He H, Sugiyama A, Snyder N, Teneche M, Liu X, Maner-Smith K, et al.
Cancer Lett
. 2023 May;
565:216210.
PMID: 37150501
Cancer cells use acetate to support the higher demand for energy and lipid biosynthesis during uncontrolled cell proliferation, as well as for acetylation of regulatory proteins. Acyl-CoA thioesterase 12 (Acot12)...