Peter D Adams
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Explore the profile of Peter D Adams including associated specialties, affiliations and a list of published articles.
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141
Citations
12922
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Recent Articles
1.
Miller K, Li B, Pierce-Hoffman H, Patel S, Lei X, Rajesh A, et al.
Nat Commun
. 2025 Mar;
16(1):2229.
PMID: 40044657
Genomic instability and inflammation are distinct hallmarks of aging, but the connection between them is poorly understood. Here we report a mechanism directly linking genomic instability and inflammation in senescent...
2.
Gu L, Zhu Y, Nandi S, Lee M, Watari K, Bareng B, et al.
Nature
. 2025 Jan;
638(8051):E30.
PMID: 39890899
No abstract available.
3.
Varanasi S, Chen D, Liu Y, Johnson M, Miller C, Ganguly S, et al.
Science
. 2025 Jan;
387(6730):192-201.
PMID: 39787217
The metabolic landscape of cancer greatly influences antitumor immunity, yet it remains unclear how organ-specific metabolites in the tumor microenvironment influence immunosurveillance. We found that accumulation of primary conjugated and...
4.
Gu L, Zhu Y, Nandi S, Lee M, Watari K, Bareng B, et al.
Nature
. 2025 Jan;
637(8045):461-469.
PMID: 39743585
Hepatocellular carcinoma (HCC) originates from differentiated hepatocytes undergoing compensatory proliferation in livers damaged by viruses or metabolic-dysfunction-associated steatohepatitis (MASH). While increasing HCC risk, MASH triggers p53-dependent hepatocyte senescence, which we...
5.
Scortegagna M, Murad R, Bina P, Feng Y, Porritt R, Terskikh A, et al.
bioRxiv
. 2024 Nov;
PMID: 39605514
Aging is a known risk factor for melanoma, yet mechanisms underlying melanoma progression and metastasis in older populations remain largely unexplored. Among the current knowledge gaps is how aging alters...
6.
Dasgupta N, Arnold R, Equey A, Gandhi A, Adams P
NPJ Aging
. 2024 Oct;
10(1):48.
PMID: 39448585
Senescence and epigenetic alterations stand out as two well-characterized hallmarks of aging. When cells become senescent, they cease proliferation and release inflammatory molecules collectively termed the Senescence-Associated Secretory Phenotype (SASP)....
7.
Alvarez-Kuglen M, Ninomiya K, Qin H, Rodriguez D, Fiengo L, Farhy C, et al.
Nat Aging
. 2024 Aug;
4(9):1308-1327.
PMID: 39210148
For efficient, cost-effective and personalized healthcare, biomarkers that capture aspects of functional, biological aging, thus predicting disease risk and lifespan more accurately and reliably than chronological age, are essential. We...
8.
Dasgupta N, Lei X, Shi C, Arnold R, Teneche M, Miller K, et al.
Mol Cell
. 2024 Aug;
84(17):3271-3287.e8.
PMID: 39178863
Cellular senescence, a stress-induced stable proliferation arrest associated with an inflammatory senescence-associated secretory phenotype (SASP), is a cause of aging. In senescent cells, cytoplasmic chromatin fragments (CCFs) activate SASP via...
9.
Ogrodnik M, Carlos Acosta J, Adams P, dAdda di Fagagna F, Baker D, Bishop C, et al.
Cell
. 2024 Aug;
187(16):4150-4175.
PMID: 39121846
Cellular senescence is a cell fate triggered in response to stress and is characterized by stable cell-cycle arrest and a hypersecretory state. It has diverse biological roles, ranging from tissue...
10.
Byrns C, Perlegos A, Miller K, Jin Z, Carranza F, Manchandra P, et al.
Nature
. 2024 Aug;
632(8026):E8.
PMID: 39112716
No abstract available.