Sarah Mustaly-Kalimi
Overview
Explore the profile of Sarah Mustaly-Kalimi including associated specialties, affiliations and a list of published articles.
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5
Citations
122
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0
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Recent Articles
1.
Mustaly-Kalimi S, Gallegos W, Marr R, Gilman-Sachs A, Peterson D, Sekler I, et al.
Proc Natl Acad Sci U S A
. 2022 Nov;
119(49):e2211999119.
PMID: 36442130
Impairments in neural lysosomal- and autophagic-mediated degradation of cellular debris contribute to neuritic dystrophy and synaptic loss. While these are well-characterized features of neurodegenerative disorders such as Alzheimer's disease (AD),...
2.
McDaid J, Mustaly-Kalimi S, Stutzmann G
Cells
. 2020 Dec;
9(12).
PMID: 33321866
Ca homeostasis is essential for multiple neuronal functions and thus, Ca dyshomeostasis can lead to widespread impairment of cellular and synaptic signaling, subsequently contributing to dementia and Alzheimer's disease (AD)....
3.
Schrank S, McDaid J, Briggs C, Mustaly-Kalimi S, Brinks D, Houcek A, et al.
Int J Mol Sci
. 2020 Feb;
21(3).
PMID: 32033164
Traditional approaches to studying Alzheimer's disease (AD) using mouse models and cell lines have advanced our understanding of AD pathogenesis. However, with the growing divide between model systems and clinical...
4.
Chakroborty S, Hill E, Christian D, Helfrich R, Riley S, Schneider C, et al.
Mol Neurodegener
. 2019 Jan;
14(1):7.
PMID: 30670054
Background: Identifying effective strategies to prevent memory loss in AD has eluded researchers to date, and likely reflects insufficient understanding of early pathogenic mechanisms directly affecting memory encoding. As synaptic...
5.
Calcium Signaling Deficits in Glia and Autophagic Pathways Contributing to Neurodegenerative Disease
Mustaly-Kalimi S, Littlefield A, Stutzmann G
Antioxid Redox Signal
. 2018 Apr;
29(12):1158-1175.
PMID: 29634342
Significance: Numerous cellular processes and signaling mechanisms have been identified that contribute to Alzheimer's disease (AD) pathology; however, a comprehensive or unifying pathway that binds together the major disease features...