Samantha L Sampson
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Explore the profile of Samantha L Sampson including associated specialties, affiliations and a list of published articles.
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44
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1043
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Recent Articles
11.
Paarwater B, Mouton J, Sampson S, Malherbe S, Shaw J, Walzl G, et al.
Am J Physiol Lung Cell Mol Physiol
. 2021 Jul;
321(3):L566-L575.
PMID: 34287085
The influence of smoke-derived or air pollution-derived cytoplasmic particulate matter (PM) can be detrimental and can lead to failed lung immunity. We investigated mycobacterial uptake, intracellular replication, and soluble immune-mediator...
12.
Gallant J, Heunis T, Beltran C, Schildermans K, Bruijns S, Mertens I, et al.
Front Immunol
. 2021 Jul;
12:702359.
PMID: 34276695
It was previously shown that secretion of PE-PGRS and PPE-MPTR proteins is abolished in clinical isolates with a deletion in the operon, which is associated with increased virulence. Here we...
13.
Kapp E, Joubert J, Sampson S, Warner D, Seldon R, Jordaan A, et al.
Adv Pharmacol Pharm Sci
. 2021 Jul;
2021:5583342.
PMID: 34240057
has developed extensive resistance to numerous antimycobacterial agents used in the treatment of tuberculosis. Insufficient intracellular accumulation of active moieties allows for selective survival of mycobacteria with drug resistance mutations...
14.
Gallant J, Mouton J, Ummels R, Ten Hagen-Jongman C, Kriel N, Pain A, et al.
NAR Genom Bioinform
. 2021 Feb;
2(2):lqaa033.
PMID: 33575588
is a facultative intracellular pathogen responsible for causing tuberculosis. The harsh environment in which survives requires this pathogen to continuously adapt in order to maintain an evolutionary advantage. However, the...
15.
Goossens S, Sampson S, Van Rie A
Clin Microbiol Rev
. 2020 Oct;
34(1).
PMID: 33055230
Successful treatment of tuberculosis (TB) can be hampered by populations that are temporarily able to survive antibiotic pressure in the absence of drug resistance-conferring mutations, a phenomenon termed drug tolerance....
16.
Parbhoo T, Sampson S, Mouton J
Cytometry A
. 2020 May;
97(7):683-693.
PMID: 32437069
The ability of the bacterial pathogen Mycobacterium tuberculosis to adapt and survive within human cells to disseminate to other individuals and cause active disease is poorly understood. Research supports that...
17.
Chirenda J, Gwitira I, Warren R, Sampson S, Murwira A, Masimirembwa C, et al.
PLoS One
. 2020 Apr;
15(4):e0231637.
PMID: 32315335
Introduction: The contribution of high tuberculosis (TB) transmission pockets in propagating area-wide transmission has not been adequately described in Zimbabwe. This study aimed to describe the presence of hotspot transmission...
18.
Kriel N, Heunis T, Sampson S, Gey van Pittius N, Williams M, Warren R
BMC Mol Cell Biol
. 2020 Apr;
21(1):19.
PMID: 32293251
Background: Transcriptional responses required to maintain cellular homeostasis or to adapt to environmental stress, is in part mediated by several nucleic-acid associated proteins. In this study, we sought to establish...
19.
Mouton J, Heunis T, Dippenaar A, Gallant J, Kleynhans L, Sampson S
Front Microbiol
. 2019 Sep;
10:1922.
PMID: 31481950
Although currently available model organisms such as and Bacillus Calmette-Guérin (BCG) have significantly contributed to our understanding of tuberculosis (TB) biology, these models have limitations such as differences in genome...
20.
Dippenaar A, De Vos M, Marx F, Adroub S, van Helden P, Pain A, et al.
Infect Genet Evol
. 2019 Jul;
75:103948.
PMID: 31276801
Recurrent tuberculosis (TB) after successful TB treatment occurs due to endogenous reactivation (relapse) or exogenous reinfection. We revisited the conclusions of relapse in a high TB incidence setting that were...