S C Cannon
Overview
Explore the profile of S C Cannon including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
46
Citations
1483
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Matthews E, Fialho D, Tan S, Venance S, Cannon S, Sternberg D, et al.
Brain
. 2009 Nov;
133(Pt 1):9-22.
PMID: 19917643
The non-dystrophic myotonias are an important group of skeletal muscle channelopathies electrophysiologically characterized by altered membrane excitability. Many distinct clinical phenotypes are now recognized and range in severity from severe...
2.
Venance S, Cannon S, Fialho D, Fontaine B, Hanna M, Ptacek L, et al.
Brain
. 2005 Oct;
129(Pt 1):8-17.
PMID: 16195244
Periodic paralyses (PPs) are rare inherited channelopathies that manifest as abnormal, often potassium (K)-sensitive, muscle membrane excitability leading to episodic flaccid paralysis. Hypokalaemic (HypoPP) and hyperkalaemic PP and Andersen-Tawil syndrome...
3.
Takahashi M, Cannon S
J Physiol
. 2001 Dec;
537(Pt 3):701-14.
PMID: 11744749
1. Over twenty different missense mutations in the alpha-subunit of the adult skeletal muscle Na(+) channel (hSkM1) have been identified as a cause of myotonia or periodic paralysis. We examined...
4.
Wu F, Takahashi M, Pegoraro E, Angelini C, Colleselli P, Cannon S, et al.
Neurology
. 2001 Apr;
56(7):878-84.
PMID: 11294924
Objective: To identify the molecular and physiologic abnormality in familial myotonia with cold sensitivity, hypertrophy, and no weakness. Background: Sodium channel mutations were previously identified as the cause of several...
5.
Ranganathan R, Cannon S, Horvitz H
Nature
. 2000 Dec;
408(6811):470-5.
PMID: 11100728
The neurotransmitter and neuromodulator serotonin (5-HT) functions by binding either to metabotropic G-protein-coupled receptors (for example, 5-HT1, 5-HT2, 5-HT4 to 5-HT7), which mediate 'slow' modulatory responses through numerous second messenger...
6.
Morrill J, Cannon S
J Gen Physiol
. 2000 Aug;
116(3):341-8.
PMID: 10962012
Skeletal muscle dihydropyridine (DHP) receptors function both as voltage-activated Ca(2+) channels and as voltage sensors for coupling membrane depolarization to release of Ca(2+) from the sarcoplasmic reticulum. In skeletal muscle,...
7.
8.
Vedantham V, Cannon S
Biophys J
. 2000 May;
78(6):2943-58.
PMID: 10827974
Mutations in segment IVS6 of voltage-gated Na(+) channels affect fast-inactivation, slow-inactivation, local anesthetic action, and batrachotoxin (BTX) action. To detect conformational changes associated with these processes, we substituted a cysteine...
9.
Cannon S
Kidney Int
. 2000 Mar;
57(3):772-9.
PMID: 10720928
Several heritable forms of myotonia and periodic paralysis are caused by missense mutations in the voltage-gated sodium channel of skeletal muscle. Mutations produce gain-of-function defects, either disrupted inactivation or enhanced...
10.
Struyk A, Scoggan K, Bulman D, Cannon S
J Neurosci
. 2000 Jan;
20(23):8610-7.
PMID: 11102465
Missense mutations of the human skeletal muscle voltage-gated Na channel (hSkM1) underlie a variety of diseases, including hyperkalemic periodic paralysis (HyperPP), paramyotonia congenita, and potassium-aggravated myotonia. Another disorder of sarcolemmal...