Ronggen Fu
Overview
Explore the profile of Ronggen Fu including associated specialties, affiliations and a list of published articles.
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8
Citations
636
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Recent Articles
1.
Gorrie G, Fecto F, Radzicki D, Weiss C, Shi Y, Dong H, et al.
Proc Natl Acad Sci U S A
. 2014 Sep;
111(40):14524-9.
PMID: 25246588
Mutations in the gene encoding ubiquilin2 (UBQLN2) cause amyotrophic lateral sclerosis (ALS), frontotemporal type of dementia, or both. However, the molecular mechanisms are unknown. Here, we show that ALS/dementia-linked UBQLN2(P497H)...
2.
Jiang M, Schuster J, Fu R, Siddique T, Heckman C
J Neurosci
. 2009 Dec;
29(48):15031-8.
PMID: 19955354
Amyotrophic lateral sclerosis (ALS) is characterized by progressive degeneration of motoneurons. One potential mechanism is excitotoxicity. We studied the behaviors of spinal neurons using an in vitro preparation of the...
3.
Zhou J, Yi J, Fu R, Liu E, Siddique T, Rios E, et al.
J Biol Chem
. 2009 Nov;
285(1):705-12.
PMID: 19889637
Amyotrophic lateral sclerosis (ALS) is a fatal neuromuscular disorder characterized by degeneration of motor neurons and atrophy of skeletal muscle. Mutations in the superoxide dismutase (SOD1) gene are linked to...
4.
Deng H, Han-Xiang D, Jiang H, Hujun J, Fu R, Ronggen F, et al.
Hum Mol Genet
. 2008 Apr;
17(15):2310-9.
PMID: 18424447
Mutations in Cu,Zn superoxide dismutase (SOD1) are associated with amyotrophic lateral sclerosis (ALS). Among more than 100 ALS-associated SOD1 mutations, premature termination codon (PTC) mutations exclusively occur in exon 5,...
5.
Deng H, Zhai H, Fu R, Shi Y, Gorrie G, Yang Y, et al.
Hum Mol Genet
. 2007 Sep;
16(23):2911-20.
PMID: 17855450
Mutations in Alsin are associated with chronic juvenile amyotrophic lateral sclerosis (ALS2), juvenile primary lateral sclerosis and infantile-onset ascending spastic paralysis. The primary pathology and pathogenic mechanism of the disease...
6.
Deng H, Shi Y, Furukawa Y, Zhai H, Fu R, Liu E, et al.
Proc Natl Acad Sci U S A
. 2006 Apr;
103(18):7142-7.
PMID: 16636275
Twenty percent of the familial form of amyotrophic lateral sclerosis (ALS) is caused by mutations in the Cu, Zn-superoxide dismutase gene (SOD1) through the gain of a toxic function. The...
7.
Furukawa Y, Fu R, Deng H, Siddique T, OHalloran T
Proc Natl Acad Sci U S A
. 2006 Apr;
103(18):7148-53.
PMID: 16636274
Point mutations in Cu, Zn-superoxide dismutase (SOD1) cause a familial form of the neurodegenerative disease amyotrophic lateral sclerosis (ALS). Aggregates of mutant SOD1 proteins are observed in histopathology and are...
8.
Kuo J, Schonewille M, Siddique T, Schults A, Fu R, Bar P, et al.
J Neurophysiol
. 2003 Oct;
91(1):571-5.
PMID: 14523070
ALS (amyotrophic lateral sclerosis) is an adult-onset and deadly neurodegenerative disease characterized by a progressive and selective loss of motoneurons. Transgenic mice overexpressing a mutated human gene (G93A) coding for...