Ron T McElmurry
Overview
Explore the profile of Ron T McElmurry including associated specialties, affiliations and a list of published articles.
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Articles
18
Citations
732
Followers
0
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Recent Articles
1.
Felices M, Lenvik T, Kodal B, Lenvik A, Hinderlie P, Bendzick L, et al.
Cancer Immunol Res
. 2020 Jul;
8(9):1139-1149.
PMID: 32661096
Natural killer (NK) cells are potent immune modulators that can quickly lyse tumor cells and elicit inflammatory responses. These characteristics make them ideal candidates for immunotherapy. However, unlike T cells,...
2.
Yellamilli A, Ren Y, McElmurry R, Lambert J, Gross P, Mohsin S, et al.
FASEB J
. 2020 Feb;
34(4):5642-5657.
PMID: 32100368
The adult mammalian heart has a limited regenerative capacity. Therefore, identification of endogenous cells and mechanisms that contribute to cardiac regeneration is essential for the development of targeted therapies. The...
3.
Xing Y, Smith M, Goetz C, McElmurry R, Parker S, Min D, et al.
J Immunol
. 2018 Oct;
201(11):3320-3328.
PMID: 30373854
Age-related thymic involution is characterized by a decrease in thymic epithelial cell (TEC) number and function parallel to a disruption in their spatial organization, resulting in defective thymocyte development and...
4.
Webber B, OConnor K, McElmurry R, Durgin E, Eide C, Lees C, et al.
Lab Invest
. 2017 Sep;
97(10):1218-1224.
PMID: 28892093
Recessive dystrophic epidermolysis bullosa (RDEB) is a debilitating and ultimately lethal blistering disease caused by mutations to the Col7a1 gene. Development of novel cell therapies for the treatment of RDEB...
5.
Webber B, Osborn M, McElroy A, Twaroski K, Lonetree C, DeFeo A, et al.
NPJ Regen Med
. 2017 Mar;
1.
PMID: 28250968
Recessive dystrophic epidermolysis bullosa (RDEB) is a severe disorder caused by mutations to the gene that deactivate production of a structural protein essential for skin integrity. Haematopoietic cell transplantation can...
6.
Osborn M, McElmurry R, Peacock B, Tolar J, Blazar B
Mol Ther
. 2017 Feb;
16(8):1459-1466.
PMID: 28182871
Mucopolysaccharidosis type I (Hurler syndrome) is caused by a deficiency of the enzyme α-l-iduronidase (IDUA), and is characterized by widespread lysosomal glycosaminoglycan (GAG) accumulation. Successful treatment of central nervous system...
7.
Modiano J, Lindborg B, McElmurry R, Lewellen M, Forster C, Zamora E, et al.
Cancer Immunol Immunother
. 2015 Aug;
64(11):1449-60.
PMID: 26250807
The potential of mesenchymal stromal cells (MSCs) to inhibit anti-tumor immunity is becoming increasingly well recognized, but the precise steps affected by these cells during the development of an anti-tumor...
8.
Tolar J, Xia L, Riddle M, Lees C, Eide C, McElmurry R, et al.
J Invest Dermatol
. 2010 Dec;
131(4):848-56.
PMID: 21124339
Recessive dystrophic epidermolysis bullosa (RDEB) is an inherited blistering skin disorder caused by mutations in the COL7A1 gene-encoding type VII collagen (Col7), the major component of anchoring fibrils at the...
9.
Osborn M, McElmurry R, Lees C, DeFeo A, Chen Z, Kay M, et al.
Mol Ther
. 2010 Nov;
19(3):450-60.
PMID: 21081900
Mucopolysaccharidosis type I (MPS I) is a lysosomal storage disease characterized by mutations to the α-L-iduronidase (IDUA) gene resulting in inactivation of the IDUA enzyme. The loss of IDUA protein...
10.
Tolar J, Park I, Xia L, Lees C, Peacock B, Webber B, et al.
Blood
. 2010 Nov;
117(3):839-47.
PMID: 21037085
Mucopolysaccharidosis type I (MPS IH; Hurler syndrome) is a congenital deficiency of α-L-iduronidase, leading to lysosomal storage of glycosaminoglycans that is ultimately fatal following an insidious onset after birth. Hematopoietic...