Rie Amamoto
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Explore the profile of Rie Amamoto including associated specialties, affiliations and a list of published articles.
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Articles
11
Citations
306
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Recent Articles
1.
Yagi M, Toshima T, Amamoto R, Do Y, Hirai H, Setoyama D, et al.
EMBO J
. 2021 Feb;
40(8):e105268.
PMID: 33528041
Mitochondrial translation dysfunction is associated with neurodegenerative and cardiovascular diseases. Cells eliminate defective mitochondria by the lysosomal machinery via autophagy. The relationship between mitochondrial translation and lysosomal function is unknown....
2.
Matsumoto T, Uchiumi T, Monji K, Yagi M, Setoyama D, Amamoto R, et al.
Oncogenesis
. 2017 Nov;
6(11):397.
PMID: 29184058
Tumor heterogeneity can be traced back to a small subset of cancer stem cells (CSCs), which can be derived from a single stem cell and show chemoresistance. Recent studies showed...
3.
Yagi M, Uchiumi T, Sagata N, Setoyama D, Amamoto R, Matsushima Y, et al.
Sci Rep
. 2017 Nov;
7(1):15131.
PMID: 29123152
Mitochondrial dysfunction is a critical step in the pathogenesis of many neurodegenerative diseases. The p32/ C1qbp gene functions as an essential RNA and protein chaperone in mitochondrial translation, and is...
4.
Saito T, Uchiumi T, Yagi M, Amamoto R, Setoyama D, Matsushima Y, et al.
Cardiovasc Res
. 2017 May;
113(10):1173-1185.
PMID: 28498888
Aims: Mitochondria are important organelles, dedicated to energy production. Mitochondrial p32/C1qbp, which functions as an RNA and protein chaperone, interacts with mitochondrial mRNA and is indispensable for mitochondrial function through...
5.
Monji K, Uchiumi T, Hoshizawa S, Yagi M, Matsumoto T, Setoyama D, et al.
Oncotarget
. 2016 Sep;
7(46):75221-75234.
PMID: 27655692
Cancer cells rewire their metabolism and mitochondrial oxidative phosphorylation (OXPHOS) to promote proliferation and maintenance. Cancer cells use multiple adaptive mechanisms in response to a hypo-nutrient environment. However, little is...
6.
Amamoto R, Uchiumi T, Yagi M, Monji K, Song Y, Oda Y, et al.
J Cancer
. 2016 Jan;
7(1):50-9.
PMID: 26722360
Background: Mitochondria play crucial roles in cell signaling events, interorganellar communication, aging, cell proliferation and apoptosis, and mitochondrial impairment has been shown to accelerate or modulate cancer progression. Ubiquitous mitochondrial...
7.
Fang J, Uchiumi T, Yagi M, Matsumoto S, Amamoto R, Takazaki S, et al.
Biosci Rep
. 2012 Dec;
33(2):e00021.
PMID: 23216091
Some mutations of the DHODH (dihydro-orotate dehydrogenase) gene lead to postaxial acrofacial dysostosis or Miller syndrome. Only DHODH is localized at mitochondria among enzymes of the de novo pyrimidine biosynthesis...
8.
Fang J, Uchiumi T, Yagi M, Matsumoto S, Amamoto R, Saito T, et al.
Biosci Rep
. 2012 Sep;
32(6):631-9.
PMID: 22967083
Miller syndrome is a recessive inherited disorder characterized by postaxial acrofacial dysostosis. It is caused by dysfunction of the DHODH (dihydroorotate dehydrogenase) gene, which encodes a key enzyme in the...
9.
Amamoto R, Yagi M, Song Y, Oda Y, Tsuneyoshi M, Naito S, et al.
Cancer Sci
. 2011 Jan;
102(3):639-47.
PMID: 21205079
Mitochondria are key organelles for ATP production and apoptosis. Therefore, impairment of mitochondria can modulate or accelerate cancer progression. p32, originally identified as a pre-mRNA splicing factor SF2/ASF-associated protein, is...
10.
Sakamaki R, Amamoto R, Mochida Y, Shinfuku N, Toyama K
Nutr J
. 2005 Nov;
4:31.
PMID: 16255785
Background: Abnormal body weight, dietary concerns, and unhealthy weight loss behaviors are increasingly being observed in young females in Japan. Our previous research has shown that the irregular lifestyles of...