Reza Izadpanah
Overview
Explore the profile of Reza Izadpanah including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
44
Citations
1282
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
11.
Yaghoubi H, Izadpanah A, Nedaei S, Akbari H, Mikaeiliagah E, Mostany R, et al.
Comb Chem High Throughput Screen
. 2020 Nov;
24(10):1714-1726.
PMID: 33208063
Background: The use of nanoparticles has markedly increased in biomedical sciences. The silver nanoparticles (AgNPs) have been investigated for their applicability to deliver chemotherapeutic/antibacterial agents to treat cancer or infections...
12.
Yoshida T, Das N, Carpenter A, Izadpanah R, Kumar S, Gautam S, et al.
Cell Signal
. 2020 Jun;
73:109690.
PMID: 32553549
Minocycline, an FDA-approved second-generation semisynthetic tetracycline, exerts antioxidant, anti-apoptotic and anti-inflammatory effects, independent of its antimicrobial properties. Interleukin (IL)-17A is an immune and inflammatory mediator, and its sustained induction is...
13.
Lin D, Ochoa J, Barabadi Z, Pfnur A, Braun S, Izadpanah R, et al.
J Stem Cells Regen Med
. 2020 Jun;
16(1):10-15.
PMID: 32536766
Tissue engineering is limited by the time of culture expansion of cells needed for scaffold seeding. Thus, a simple means of accelerated stem cell proliferation could represent a significant advance....
14.
Alt E, Worner P, Pfnur A, Ochoa J, Schachtele D, Barabadi Z, et al.
Sci Rep
. 2020 Jun;
10(1):8834.
PMID: 32483202
Here we investigated the roles of Rab27a, a player in exosome release, and TRAF3IP2, an inflammatory mediator, in development and metastasis of breast cancer (BC) in vivo. Knockdown (KD) of...
15.
Saleh F, Chandra P, Lin D, Robinson J, Izadpanah R, Mondal D, et al.
J Immunol
. 2020 Mar;
204(7):1998-2005.
PMID: 32144163
Mice have been used as accepted tools for investigating complex human diseases and new drug therapies because of their shared genetics and anatomical characteristics with humans. However, the tissues in...
16.
Mummidi S, Das N, Carpenter A, Yoshida T, Yariswamy M, Mostany R, et al.
J Cell Physiol
. 2019 May;
234(12):22242-22259.
PMID: 31074012
Sustained inflammation and matrix metalloproteinase (MMP) activation contribute to vascular occlusive/proliferative disorders. Interleukin-17 (IL-17) is a proinflammatory cytokine that signals mainly via TRAF3 Interacting Protein 2 (TRAF3IP2), an upstream regulator...
17.
Worner P, Schachtele D, Barabadi Z, Srivastav S, Chandrasekar B, Izadpanah R, et al.
Stem Cells Dev
. 2018 Dec;
28(5):341-352.
PMID: 30572805
How mesenchymal stem cells (MSCs) interact with tumor cells and promote tumor growth is not well understood. In this study, we demonstrate that when naive MSCs and malignant breast cancer...
18.
Bayat N, Izadpanah R, Ebrahimi-Barough S, Norouzi Javidan A, Ai A, Mokhtari Ardakan M, et al.
Asian Pac J Cancer Prev
. 2018 Sep;
19(9):2553-2560.
PMID: 30256055
Purpose: Glioblastoma multiform (GBM) is the most aggressive glial neoplasm. Researchers have exploited the
fact that GBMs are highly vascularized tumors. Anti-angiogenic strategies including those targeting VEGF pathway
have been...
19.
Alt E, Barabadi Z, Pfnur A, Ochoa J, Daneshimehr F, Lang L, et al.
Oncotarget
. 2018 Jul;
9(51):29772-29788.
PMID: 30038719
Glioblastoma multiforme (glioblastoma) remains one of the deadliest cancers. Pro-inflammatory and pro-tumorigenic mediators present in tumor microenvironment (TME) facilitate communication between tumor cells and adjacent non-malignant cells, resulting in glioblastoma...
20.
Das N, Carpenter A, Yoshida T, Kumar S, Gautam S, Mostany R, et al.
J Mol Cell Cardiol
. 2018 Jul;
121:107-123.
PMID: 29981796
Persistent inflammation promotes development and progression of heart failure (HF). TWEAK (TNF-Related WEAK Inducer Of Apoptosis), a NF-κB- and/or AP-1-responsive proinflammatory cytokine that signals via TWEAK receptor (TWEAKR), is expressed...