R C Venema
Overview
Explore the profile of R C Venema including associated specialties, affiliations and a list of published articles.
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Articles
46
Citations
1408
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Recent Articles
1.
Harris M, Ju H, Venema V, BLACKSTONE M, Venema R
Gen Pharmacol
. 2001 Dec;
35(3):165-70.
PMID: 11744239
Previously we described ENAP-1, a 90-kDa protein that is tyrosine-phosphorylated in endothelial cells in response to bradykinin (BK) stimulation and is associated with endothelial nitric oxide synthase (eNOS). Subsequently, other...
2.
Harris M, Ju H, Venema V, Liang H, Zou R, Michell B, et al.
J Biol Chem
. 2001 May;
276(19):16587-91.
PMID: 11340086
Endothelial nitric-oxide synthase (eNOS) is phosphorylated at Ser-1179 (bovine sequence) by Akt after growth factor or shear stress stimulation of endothelial cells, resulting in increased eNOS activity. Purified eNOS is...
3.
Ju H, Venema V, Liang H, Harris M, Zou R, Venema R
Biochem J
. 2000 Sep;
351(Pt 1):257-64.
PMID: 10998369
Bradykinin (BK) is an important physiological regulator of endothelial cell function. In the present study, we have examined the role of the Janus-activated kinase (JAK)/signal transducers and activators of transcription...
4.
Bartoli M, Gu X, Tsai N, Venema R, Brooks S, Marrero M, et al.
J Biol Chem
. 2000 Aug;
275(43):33189-92.
PMID: 10961983
Vascular endothelial growth factor (VEGF) intracellular signaling in endothelial cells is initiated by the activation of distinct tyrosine kinase receptors, VEGFR1 (Flt-1) and VEGFR2 (Flk-1/KDR). Because the tyrosine kinase-dependent transcription...
5.
Huang A, Vita J, Venema R, Keaney Jr J
J Biol Chem
. 2000 Apr;
275(23):17399-406.
PMID: 10749876
Ascorbic acid enhances NO bioactivity in patients with vascular disease through unclear mechanism(s). We investigated the role of intracellular ascorbic acid in endothelium-derived NO bioactivity. Incubation of porcine aortic endothelial...
6.
Golser R, Gorren A, Leber A, Andrew P, Habisch H, Werner E, et al.
J Biol Chem
. 2000 Feb;
275(8):5291-6.
PMID: 10681501
Endothelial nitric-oxide synthase (type III) (eNOS) was reported to form an inhibitory complex with the bradykinin receptor B2 (B2R) from which the enzyme is released in an active form upon...
7.
Amiri F, Venema V, Wang X, Ju H, Venema R, Marrero M
J Biol Chem
. 1999 Nov;
274(45):32382-6.
PMID: 10542280
We have shown previously that angiotensin II (Ang II) activates the janus-activated kinase (JAK)/signal transducers and activators of transcription (STAT) pathway in vascular smooth muscle cells (VSMCs) and that activation...
8.
Marrero M, Venema V, Ju H, He H, Liang H, Caldwell R, et al.
Biochem J
. 1999 Oct;
343 Pt 2:335-40.
PMID: 10510297
The endothelial nitric oxide synthase (eNOS) is activated in response to stimulation of endothelial cells by a number of vasoactive substances including, bradykinin (BK), angiotensin II (Ang II), endothelin-1 (ET-1)...
9.
He H, Venema V, Gu X, Venema R, Marrero M, Caldwell R
J Biol Chem
. 1999 Aug;
274(35):25130-5.
PMID: 10455194
Vascular endothelial growth factor (VEGF) is a potent endothelial cell-specific mitogen that promotes angiogenesis, vascular hyperpermeability, and vasodilation by autocrine mechanisms involving nitric oxide (NO) and prostacyclin (PGI(2)) production. These...
10.
Liang H, Venema V, Wang X, Ju H, Venema R, Marrero M
J Biol Chem
. 1999 Jul;
274(28):19846-51.
PMID: 10391929
Ligand binding to the angiotensin II (Ang II) AT1 receptor on vascular smooth muscle cells (VSMCs) activates the Janus-activated kinase (JAK)/signal transducers and activators of transcription (STAT) pathway. We have...