R A Eisenberg
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Explore the profile of R A Eisenberg including associated specialties, affiliations and a list of published articles.
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125
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2621
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Recent Articles
1.
Freed J, Marrs A, Vanderwall J, Cohen P, Eisenberg R
J Immunol
. 2000 Apr;
164(9):4697-705.
PMID: 10779775
The systemic lupus erythematosus-like syndrome in MRL/lpr mice involves high-titered IgG autoantibodies, particularly antinuclear Abs that target histones, DNA, and RNA particles. Although T cell help is required for the...
2.
Feuerstein N, Chen F, Madaio M, Maldonado M, Eisenberg R
J Immunol
. 1999 Nov;
163(10):5287-97.
PMID: 10553051
Abrogation of peripheral tolerance in transgenic mice that express a uniform B-cell receptor may create a powerful tool to examine the molecular mechanisms that underlie the autoimmune response in B...
3.
Maldonado M, MacDonald G, Kakkanaiah V, Fecho K, Dransfield M, Sekiguchi D, et al.
J Immunol
. 1999 Sep;
163(6):3138-42.
PMID: 10477580
We have previously shown that the gld autoimmune syndrome is suppressed in lethally irradiated gld mice reconstituted with a mixture of normal and gld bone marrow (BM). Furthermore, in vivo...
4.
Hoch S, Eisenberg R, Sharp G
Clin Immunol
. 1999 Aug;
92(2):203-8.
PMID: 10444365
Anti-Sm antibodies are intrinsically associated with systemic lupus erythematosus. The major targets are the so-called B and D polypeptides. The number of Sm targets increased upon the report that SDS-PAGE...
5.
Maldonado M, Kakkanaiah V, MacDonald G, Chen F, Reap E, Balish E, et al.
J Immunol
. 1999 Jun;
162(11):6322-30.
PMID: 10352243
It has been proposed that the "normal" stimulation of the immune system that occurs from interactions with environmental stimuli, whether infectious or dietary, is necessary for the initiation and/or continuation...
6.
Chen F, Maldonado M, Madaio M, Eisenberg R
J Immunol
. 1998 Dec;
161(11):5880-5.
PMID: 9834067
Chronic graft-vs-host (cGVH) disease induced by the transfer of Ia-incompatible spleen cells from one normal mouse strain (such as B6.C-H2(bm12)/KhEg (bm12)) to another (such as C57BL/6) causes an autoimmune syndrome...
7.
Weintraub J, Godfrey V, Wolthusen P, Cheek R, Eisenberg R, Cohen P
Cell Immunol
. 1998 Jun;
186(1):8-17.
PMID: 9637760
The lpr mutation in mice results in premature termination of transcription of the gene encoding the apoptosis-signaling receptor Fas. As a result, lpr mice develop severe lymphoproliferation and lupus-like autoantibodies....
8.
Sobel E, Kakkanaiah V, Schiffenbauer J, Reap E, Cohen P, Eisenberg R
J Immunol
. 1998 May;
160(3):1497-503.
PMID: 9570572
lpr, a murine mutation of the Fas apoptosis receptor, causes lymphadenopathy and autoantibody production, with lymphadenopathy primarily due to a population of CD4-CD8-B220+ T cells. Previous in vivo experiments, in...
9.
Weintraub J, Eisenberg R, Cohen P
J Immunol
. 1997 Oct;
159(8):4117-26.
PMID: 9379003
C57BL/6-gld/gld (B6/gld) mice have a point mutation in the gene for Fas ligand (FasL) resulting in nonfunctional FasL protein. We hypothesized that the lack of normal Fas/FasL interactions in these...
10.
Kakkanaiah V, Sobel E, MacDonald G, Cheek R, Cohen P, Eisenberg R
J Immunol
. 1997 Jul;
159(2):1027-35.
PMID: 9218626
Anti-Sm Abs are specific markers of human systemic lupus erythematosus (SLE) and of murine models of this disease. In humans, anti-Sm Abs are mostly IgG1, and in MRL/lpr mice, IgG2a;...