Peter Holmans
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Explore the profile of Peter Holmans including associated specialties, affiliations and a list of published articles.
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156
Citations
10758
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Recent Articles
1.
Barnett E, Hess J, Hou J, Escott-Price V, Fennema-Notestine C, Kremen W, et al.
medRxiv
. 2025 Mar;
PMID: 40061341
Background: Genetic risk factors for psychiatric and neurodegenerative disorders are well documented. However, some individuals with high genetic risk remain unaffected, and the mechanisms underlying such resilience remain poorly understood....
2.
Hou J, Hess J, Zhang C, van Rooij J, Hearn G, Fan C, et al.
Am J Med Genet B Neuropsychiatr Genet
. 2024 Dec;
:e33019.
PMID: 39679839
The comprehensive genome-wide nature of transcriptome studies in Alzheimer's disease (AD) should provide a reliable description of disease molecular states. However, the genes and molecular systems nominated by transcriptomic studies...
3.
Smart S, Legge S, Fenner E, Pardinas A, Woolway G, Lynham A, et al.
medRxiv
. 2024 Oct;
PMID: 39371177
The missense SNP NC_000004.12:g.102267552C>T (SLC39A8.p.(Ala391Thr), rs13107325) in , which encodes a zinc transporter, has been linked to schizophrenia and is the likely causal variant for one of the genome-wide association...
4.
Lee J, McLean Z, Correia K, Shin J, Lee S, Jang J, et al.
bioRxiv
. 2024 Jul;
PMID: 38948755
Huntington's disease (HD), due to expansion of a CAG repeat in , is representative of a growing number of disorders involving somatically unstable short tandem repeats. We find that overlapping...
5.
Hall J, Chawner S, Wolstencroft J, Skuse D, Hall J, Holmans P, et al.
Transl Psychiatry
. 2024 Jun;
14(1):259.
PMID: 38890284
A range of rare mutations involving micro-deletion or -duplication of genetic material (copy number variants (CNVs)) have been associated with high neurodevelopmental and psychiatric risk (ND-CNVs). Irritability is frequently observed...
6.
Kim K, Hong E, Lee Y, McLean Z, Elezi E, Lee R, et al.
Proc Natl Acad Sci U S A
. 2024 Apr;
121(16):e2322924121.
PMID: 38607933
Many Mendelian disorders, such as Huntington's disease (HD) and spinocerebellar ataxias, arise from expansions of CAG trinucleotide repeats. Despite the clear genetic causes, additional genetic factors may influence the rate...
7.
Legge S, Pardinas A, Woolway G, Rees E, Cardno A, Escott-Price V, et al.
JAMA Psychiatry
. 2024 Mar;
81(7):681-690.
PMID: 38536179
Importance: Large-scale biobanks provide important opportunities for mental health research, but selection biases raise questions regarding the comparability of individuals with those in clinical research settings. Objective: To compare the...
8.
Hong E, Ramos E, Aziz N, Massey T, McAllister B, Lobanov S, et al.
Brain Commun
. 2024 Mar;
6(2):fcae016.
PMID: 38449714
Expansions of glutamine-coding CAG trinucleotide repeats cause a number of neurodegenerative diseases, including Huntington's disease and several of spinocerebellar ataxias. In general, age-at-onset of the polyglutamine diseases is inversely correlated...
9.
Uzochukwu E, Harding K, Hrastelj J, Kreft K, Holmans P, Robertson N, et al.
Neuroepidemiology
. 2024 Feb;
58(3):218-226.
PMID: 38377969
Objectives: The objective of this study was to model multiple sclerosis (MS) disease progression and compare disease trajectories by sex, age of onset, and year of diagnosis. Study Design And...
10.
Hall J, Chawner S, Wolstencroft J, Skuse D, Holmans P, Owen M, et al.
medRxiv
. 2023 Dec;
PMID: 38106165
Background: A range of rare mutations involving micro-deletion or -duplication of genetic material (copy number variants (CNVs)) have been associated with high neurodevelopmental and psychiatric risk (ND-CNVs). Irritability is frequently...