P R Craddock
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Explore the profile of P R Craddock including associated specialties, affiliations and a list of published articles.
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44
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1411
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Recent Articles
11.
Vercellotti G, Hammerschmidt D, Craddock P, JACOB H
Blood
. 1982 Jun;
59(6):1299-1304.
PMID: 7082830
Perfluorocarbons have shown promise as clinical blood substitutes. Although early experience in Japan with one such product--Fluosol-DA--has been uncomplicated, we observed an adverse pulmonary reaction in the first American patient...
12.
Schmeling D, Gemmell C, Craddock P, Quie P, Peterson P
Inflammation
. 1981 Dec;
5(4):313-22.
PMID: 7327697
The effect of staphylococcal alpha-toxin on the chemotactic response of human polymorphonuclear leukocytes was studied by measuring the migration of alpha-toxin-treated cells either through membrane filters toward C5a or under...
13.
Weisdorf D, Hammerschmidt D, JACOB H, Craddock P
J Lab Clin Med
. 1981 Dec;
98(6):823-30.
PMID: 7310225
C5ades arg is an important mediator of the tissue injury associated with intravascular complement activation and exerts its effects by causing granulocyte aggregation, leukoembolism, and oxidative damage to endothelial cells....
14.
Skubitz K, Craddock P, Hammerschmidt D, August J
J Clin Invest
. 1981 Jul;
68(1):13-20.
PMID: 7251857
Inhibition of complement-mediated granulocyte aggregation has recently been proposed as a mechanism of action of high-dose corticosteroids in shock states. Postulating that such inhibition might be effected through alteration of...
15.
Hammerschmidt D, Greenberg C, Yamada O, Craddock P, JACOB H
J Lab Clin Med
. 1981 Jul;
98(1):68-77.
PMID: 6265570
Previous studies in experimental myocardial infarction have suggested that PMNs and plasma C might interact to intensify ischemic injury. From the finding of large amounts of activated C (specifically C5a)...
16.
Deleterious effects of endotoxin on cultured endothelial cells: an in vitro model of vascular injury
Yamada O, MOLDOW C, Sacks T, Craddock P, Boogaerts M, JACOB H
Inflammation
. 1981 Jun;
5(2):115-26.
PMID: 6268539
The effects of endotoxin-triggered granulocytes on the viability of endothelial cells in vitro was investigated. Endotoxin or its lipid A component caused granulocytes to adhere to and significantly damage cultured...
17.
Skubitz K, Craddock P
J Clin Invest
. 1981 May;
67(5):1383-91.
PMID: 7229031
The transient granulocytopenia of hemodialysis results indirectly from plasma complement activation by dialyzer cellophane membranes. The C5a(desarg) so produced can induce reversible granulocyte aggregation in vitro and in vivo, and...
18.
Hammerschmidt D, Stroncek D, Bowers T, Lammi-Keefe C, KURTH D, Ozalins A, et al.
J Thorac Cardiovasc Surg
. 1981 Mar;
81(3):370-7.
PMID: 6970304
Complement activation and pulmonary leukostasis with neutropenia occur in hemodialysis and filtration leukapheresis, with attendant pulmonary dysfunction. Wondering whether similar phenomena might attend cardiopulmonary bypass (CPB), we studied 34 patients...
19.
Hammerschmidt D, Harris P, Wayland J, Craddock P, JACOB H
Am J Pathol
. 1981 Feb;
102(2):146-50.
PMID: 7468764
Previous studies from our laboratories have demonstrated that granulocytes (PMNs), when exposed to activated complement (C) (specifically C5a), will aggregate and be provoked to damage cultured endothelial cells in vitro;...
20.
JACOB H, Goldstein I, Shapiro I, Craddock P, Hammerschmidt D, Weissmann G
Arch Intern Med
. 1981 Jan;
141(1):134-6.
PMID: 7447575
During an episode of acute alcoholic pancreatitis, severe visual loss and the funduscopic appearance of Purtscher's retinopathy-a syndrome thought to be caused by posterior retinal microembolization-developed in a patient. We...