Othon L Gervasio
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Explore the profile of Othon L Gervasio including associated specialties, affiliations and a list of published articles.
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14
Citations
614
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Recent Articles
1.
Ho P, Hiwase D, Ramakrishna R, Viiala N, Solterbeck A, Traficante R, et al.
Hemasphere
. 2019 Nov;
3(3):e224.
PMID: 31723837
The significant morbidity and mortality associated with iron overload can be reduced by effective iron chelation. Magnetic resonance imaging (MRI) provides accurate and reproducible iron load assessment. The aim of...
2.
Ho P, Tay L, Teo J, Marlton P, Grigg A, St Pierre T, et al.
Eur J Haematol
. 2016 Aug;
98(2):97-105.
PMID: 27537786
Objectives: To assess the effect of iron chelation therapy with deferasirox on cardiac iron and function in patients with transfusion-dependent thalassemia major, sickle cell disease (SCD), and myelodysplastic syndromes (MDS)....
3.
Zhang B, Whitehead N, Gervasio O, Reardon T, Vale M, Fatkin D, et al.
J Appl Physiol (1985)
. 2012 Mar;
112(12):2077-86.
PMID: 22461447
Muscles that are stretched during contraction (eccentric contractions) show deficits in force production and a variety of structural changes, including loss of antibody staining of cytoskeletal proteins. Extracellular Ca(2+) entry...
4.
Gervasio O, Phillips W, Cole L, Allen D
J Cell Sci
. 2011 Nov;
124(Pt 21):3581-90.
PMID: 22045729
Caveolae are invaginations of the plasma membrane that are formed by caveolins. Caveolar membranes are also enriched in cholesterol, glycosphingolipids and signaling enzymes such as Src kinase. Here we investigate...
5.
Allen D, Gervasio O, Yeung E, Whitehead N
Can J Physiol Pharmacol
. 2010 Mar;
88(2):83-91.
PMID: 20237582
Duchenne muscular dystrophy (DMD) is a severe muscle-wasting disease caused by the absence of the cytoskeletal protein dystrophin. Experiments on the mdx mouse, a model of DMD, have shown that...
6.
Whiteman I, Gervasio O, Cullen K, Guillemin G, Jeong E, Witting P, et al.
J Neurosci
. 2009 Oct;
29(41):12994-3005.
PMID: 19828813
In Alzheimer's disease (AD), rod-like cofilin aggregates (cofilin-actin rods) and thread-like inclusions containing phosphorylated microtubule-associated protein (pMAP) tau form in the brain (neuropil threads), and the extent of their presence...
7.
Gervasio O, Whitehead N, Yeung E, Phillips W, Allen D
J Cell Sci
. 2008 Jun;
121(Pt 13):2246-55.
PMID: 18544631
Transient receptor potential canonical 1 (TRPC1), a widely expressed calcium (Ca(2+))-permeable channel, is potentially involved in the pathogenesis of Duchenne muscular dystrophy (DMD). Ca(2+) influx through stretch-activated channels, possibly formed...
8.
Brockhausen J, Cole R, Gervasio O, Ngo S, Noakes P, Phillips W
Dev Neurobiol
. 2008 May;
68(9):1153-69.
PMID: 18506821
Fluorescence resonance energy transfer (FRET) experiments at neuromuscular junctions in the mouse tibialis anterior muscle show that postsynaptic acetylcholine receptors (AChRs) become more tightly packed during the first month of...
9.
Cole R, Reddel S, Gervasio O, Phillips W
Ann Neurol
. 2008 Apr;
63(6):782-9.
PMID: 18384168
Objective: A subset of myasthenia gravis patients that are seronegative for anti-acetylcholine receptor (anti-AChR) antibodies are instead seropositive for antibodies against the muscle-specific kinase (anti-MuSK-positive). Here, we test whether transfer...
10.
Whitehead N, Pham C, Gervasio O, Allen D
J Physiol
. 2008 Feb;
586(7):2003-14.
PMID: 18258657
Duchenne muscular dystrophy (DMD) is a severe degenerative muscle disease caused by a mutation in the gene encoding dystrophin, a protein linking the cytoskeleton to the extracellular matrix. In this...