Olga Sthandier
Overview
Explore the profile of Olga Sthandier including associated specialties, affiliations and a list of published articles.
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Articles
17
Citations
3198
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Recent Articles
1.
Legnini I, Di Timoteo G, Rossi F, Morlando M, Briganti F, Sthandier O, et al.
Mol Cell
. 2017 Mar;
66(1):22-37.e9.
PMID: 28344082
Circular RNAs (circRNAs) constitute a family of transcripts with unique structures and still largely unknown functions. Their biogenesis, which proceeds via a back-splicing reaction, is fairly well characterized, whereas their...
2.
Cazzella V, Martone J, Pinnaro C, Santini T, Twayana S, Sthandier O, et al.
Mol Ther
. 2012 Sep;
20(11):2134-42.
PMID: 22968481
Exon skipping has been demonstrated to be a successful strategy for the gene therapy of Duchenne muscular dystrophy (DMD): the rational being to convert severe Duchenne forms into milder Becker...
3.
Cesana M, Cacchiarelli D, Legnini I, Santini T, Sthandier O, Chinappi M, et al.
Cell
. 2011 Oct;
147(2):358-69.
PMID: 22000014
Recently, a new regulatory circuitry has been identified in which RNAs can crosstalk with each other by competing for shared microRNAs. Such competing endogenous RNAs (ceRNAs) regulate the distribution of...
4.
Cacchiarelli D, Incitti T, Martone J, Cesana M, Cazzella V, Santini T, et al.
EMBO Rep
. 2011 Jan;
12(2):136-41.
PMID: 21212803
Duchenne muscular dystrophy (DMD)--which is caused by mutations in the dystrophin gene-is one of the most severe myopathies. Among therapeutic strategies, exon skipping allows the rescue of dystrophin synthesis through...
5.
Cacchiarelli D, Martone J, Girardi E, Cesana M, Incitti T, Morlando M, et al.
Cell Metab
. 2010 Aug;
12(4):341-351.
PMID: 20727829
In Duchenne muscular dystrophy (DMD) the absence of dystrophin at the sarcolemma delocalizes and downregulates nitric oxide synthase (nNOS); this alters S-nitrosylation of HDAC2 and its chromatin association. We show...
6.
Incitti T, De Angelis F, Cazzella V, Sthandier O, Pinnaro C, Legnini I, et al.
Mol Ther
. 2010 Jun;
18(9):1675-82.
PMID: 20551908
One promising approach for the gene therapy of Duchenne muscular dystrophy (DMD) is exon skipping. When thinking of possible intervention on human, it is very crucial to identify the most...
7.
Guastafierro T, Cecchinelli B, Zampieri M, Reale A, Riggio G, Sthandier O, et al.
J Biol Chem
. 2008 Jun;
283(32):21873-80.
PMID: 18539602
Our previous data have shown that in L929 mouse fibroblasts the control of methylation pattern depends in part on poly(ADP-ribosyl)ation and that ADP-ribose polymers (PARs), both present on poly(ADP-ribosyl)ated PARP-1...
8.
Denti M, Incitti T, Sthandier O, Nicoletti C, De Angelis F, Rizzuto E, et al.
Hum Gene Ther
. 2008 May;
19(6):601-8.
PMID: 18500943
Many mutations and deletions in the dystrophin gene, responsible for Duchenne muscular dystrophy (DMD), can be corrected at the posttranscriptional level by skipping specific exons. Here we show that long-term...
9.
Caruso M, Busanello A, Sthandier O, Cavaldesi M, Gentile M, Garcia M, et al.
J Mol Biol
. 2007 Jan;
367(1):54-64.
PMID: 17239397
The first contact of a virus with the host cell surface and further entry are important steps for a successful outcome of the infection process and for the virus-associated pathogenicity....
10.
Carbone M, Reale A, Di Sauro A, Sthandier O, Garcia M, Maione R, et al.
J Mol Biol
. 2006 Sep;
363(4):773-85.
PMID: 16979186
Poly(ADP-ribose)polymerases are involved in fundamental cellular events as well as they seem to be associated to some viral infection process. In this work, the poly(ADP-ribose)polymerase-1 (PARP-1) role in the polyomavirus...