Nicholas K Foreman
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Explore the profile of Nicholas K Foreman including associated specialties, affiliations and a list of published articles.
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156
Citations
4977
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Recent Articles
11.
DeSisto J, Donson A, Griesinger A, Fu R, Riemondy K, Mulcahy Levy J, et al.
Neuro Oncol
. 2023 Nov;
26(3):538-552.
PMID: 37934854
Background: Pediatric high-grade gliomas (PHGG) are aggressive brain tumors with 5-year survival rates ranging from <2% to 20% depending upon subtype. PHGG presents differently from patient to patient and is...
12.
Grob S, Miller K, Sanford B, Donson A, Jones K, Griesinger A, et al.
J Neurooncol
. 2023 Oct;
165(1):161-169.
PMID: 37878192
Background: Neurocognitive deficits are common in pediatric brain tumor survivors. The use of single nucleotide polymorphism (SNP) analysis in DNA repair genes may identify children treated with radiation therapy for...
13.
Amani V, Riemondy K, Fu R, Griesinger A, Grimaldo E, de Sousa G, et al.
Acta Neuropathol Commun
. 2023 Sep;
11(1):158.
PMID: 37770931
Plexiform neurofibroma (PN) is a leading cause of morbidity in children with the genetic condition Neurofibromatosis Type 1 (NF1), often disfiguring or threatening vital structures. During formation of PN, a...
14.
Griesinger A, Riemondy K, Eswaran N, Donson A, Willard N, Prince E, et al.
iScience
. 2023 Sep;
26(9):107585.
PMID: 37694144
Ependymoma (EPN) is a devastating childhood brain tumor. Single-cell analyses have illustrated the cellular heterogeneity of EPN tumors, identifying multiple neoplastic cell states including a mesenchymal-differentiated subpopulation which characterizes the...
15.
Grob S, Miller K, Sanford B, Donson A, Jones K, Griesinger A, et al.
Res Sq
. 2023 Aug;
PMID: 37609195
Purpose: Neurocognitive deficits are common in pediatric brain tumor survivors. The use of single nucleotide polymorphism (SNP) analysis in DNA repair genes may identify children treated with radiation therapy for...
16.
Okonechnikov K, Joshi P, Sepp M, Leiss K, Sarropoulos I, Murat F, et al.
Neuro Oncol
. 2023 Aug;
25(10):1895-1909.
PMID: 37534924
Background: Distinguishing the cellular origins of childhood brain tumors is key for understanding tumor initiation and identifying lineage-restricted, tumor-specific therapeutic targets. Previous strategies to map the cell-of-origin typically involved comparing...
17.
Donson A, Bertrand K, Riemondy K, Gao D, Zhuang Y, Sanford B, et al.
Neuro Oncol
. 2023 May;
25(10):1854-1867.
PMID: 37246777
Background: Ependymoma (EPN) posterior fossa group A (PFA) has the highest rate of recurrence and the worst prognosis of all EPN molecular groups. At relapse, it is typically incurable even...
18.
de Sousa G, Salomao K, Nagano L, Riemondy K, Chagas P, Veronez L, et al.
Cancer Gene Ther
. 2023 Apr;
30(8):1105-1113.
PMID: 37041276
Members of the HDAC family are predictive biomarkers and regulate the tumorigenesis in several cancers. However, the role of these genes in the biology of intracranial ependymomas (EPNs) remains unexplored....
19.
Dunkel I, Doz F, Foreman N, Hargrave D, Lassaletta A, Andre N, et al.
Neuro Oncol
. 2023 Feb;
25(8):1530-1545.
PMID: 36808285
Background: Therapeutic options are limited in pediatric CNS malignancies. CheckMate 908 (NCT03130959) is an open-label, sequential-arm, phase 1b/2 study investigating nivolumab (NIVO) and NIVO + ipilimumab (IPI) in pediatric patients...
20.
Walker F, Sobral L, Danis E, Sanford B, Balakrishnan I, Wang D, et al.
bioRxiv
. 2023 Feb;
PMID: 36747867
Dynamic regulation of gene expression is fundamental for cellular adaptation to exogenous stressors. PTEFb-mediated pause-release of RNA polymerase II (Pol II) is a conserved regulatory mechanism for synchronous transcriptional induction...