Nahuai Badiola
Overview
Explore the profile of Nahuai Badiola including associated specialties, affiliations and a list of published articles.
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Articles
13
Citations
406
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0
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Recent Articles
1.
Capdevila-Busquets E, Badiola N, Arroyo R, Alcalde V, Soler-Lopez M, Aloy P
PLoS One
. 2015 Jan;
10(1):e0115352.
PMID: 25590583
A key element to delineate the biology of individual tumors is the regulation of apoptosis. In this work, we functionally characterize two breast cancer associated genes, the proteasome 26S subunit...
2.
Suarez-Calvet M, Belbin O, Pera M, Badiola N, Magrane J, Guardia-Laguarta C, et al.
J Neurochem
. 2013 Oct;
128(2):330-9.
PMID: 24117942
Autosomal-dominant Alzheimer's disease (ADAD) is a genetic disorder caused by mutations in Amyloid Precursor Protein (APP) or Presenilin (PSEN) genes. Studying the mechanisms underlying these mutations can provide insight into...
3.
Badiola N, Alcalde V, Pujol A, Munter L, Multhaup G, Lleo A, et al.
PLoS One
. 2013 Mar;
8(3):e58837.
PMID: 23520537
A key event in the pathogenesis of Alzheimer's disease (AD) is the accumulation of amyloid-β (Aβ) species in the brain, derived from the sequential cleavage of the amyloid precursor protein...
4.
Pera M, Alcolea D, Sanchez-Valle R, Guardia-Laguarta C, Colom-Cadena M, Badiola N, et al.
Acta Neuropathol
. 2012 Dec;
125(2):201-13.
PMID: 23224319
Autosomal-dominant Alzheimer disease (ADAD) is a genetic disorder caused by mutations in Amyloid Precursor Protein (APP) or Presenilin (PSEN) genes. Studies from families with ADAD have been critical to support...
5.
Soler-Lopez M, Badiola N, Zanzoni A, Aloy P
Bioessays
. 2012 Apr;
34(7):532-41.
PMID: 22513506
Here we postulate that the adapter protein evolutionarily conserved signalling intermediate in Toll pathway (ECSIT) might act as a molecular sensor in the pathogenesis of Alzheimer's disease (AD). Based on...
6.
Barneda-Zahonero B, Servitja J, Badiola N, Minano-Molina A, Fado R, Saura C, et al.
J Biol Chem
. 2012 Feb;
287(14):11351-62.
PMID: 22294685
NMDA receptor (NMDAR) stimulation promotes neuronal survival during brain development. Cerebellar granule cells (CGCs) need NMDAR stimulation to survive and develop. These neurons differentiate and mature during its migration from...
7.
Badiola N, de Oliveira R, Herrera F, Guardia-Laguarta C, Goncalves S, Pera M, et al.
PLoS One
. 2011 Nov;
6(10):e26609.
PMID: 22039514
Background: The simultaneous accumulation of different misfolded proteins in the central nervous system is a common feature in many neurodegenerative diseases. In most cases, co-occurrence of abnormal deposited proteins is...
8.
Badiola N, Suarez-Calvet M, Lleo A
CNS Neurol Disord Drug Targets
. 2010 Oct;
9(6):727-40.
PMID: 20942789
Tauopathies are neurodegenerative diseases characterized by insoluble hyperphosphorylated deposits of the microtubule-associated protein tau in the central nervous system. In these disorders, tau is believed to cause neurodegeneration and neuronal...
9.
Barneda-Zahonero B, Minano-Molina A, Badiola N, Fado R, Xifro X, Saura C, et al.
Mol Biol Cell
. 2009 Oct;
20(24):5051-63.
PMID: 19846661
Bone morphogenetic proteins (BMPs) have been implicated in the generation and postnatal differentiation of cerebellar granule cells (CGCs). Here, we examined the eventual role of BMPs on the survival of...
10.
Badiola N, Malagelada C, Llecha N, Hidalgo J, Comella J, Sabria J, et al.
Neurobiol Dis
. 2009 Jun;
35(3):438-47.
PMID: 19555759
TNF-alpha has been reported to be relevant in stroke-induced neuronal death. However the precise function of TNF-alpha in brain ischemia remains controversial since there are data supporting either a detrimental...