Mryanda J Sopel
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Explore the profile of Mryanda J Sopel including associated specialties, affiliations and a list of published articles.
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5
Citations
94
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Recent Articles
1.
Rosin N, Sopel M, Falkenham A, Lee T, Legare J
Am J Pathol
. 2015 Feb;
185(3):631-42.
PMID: 25701883
Heart failure, the leading cause of hospitalization of elderly patients, is correlated with myocardial fibrosis (ie, deposition of excess extracellular matrix proteins such as collagen). A key regulator of collagen...
2.
Rosin N, Gareau A, Betsch D, Falkenham A, Sopel M, Lee T, et al.
Fibrogenesis Tissue Repair
. 2014 Apr;
7:6.
PMID: 24721281
Background: Myocardial fibrosis is a pathological process that is characterized by disrupted regulation of extracellular matrix proteins resulting in permanent scarring of the heart tissue and eventual diastolic heart failure....
3.
Rosin N, Falkenham A, Sopel M, Lee T, Legare J
Am J Pathol
. 2013 Jan;
182(3):714-26.
PMID: 23287510
Exposure of rodents to angiotensin II (AngII) is a common model of fibrosis. We have previously shown that cellular infiltration of bone marrow-derived progenitor cells (fibrocytes) occurs before deposition of...
4.
Sopel M, Rosin N, Falkenham A, Bezuhly M, Esmon C, Lee T, et al.
PLoS One
. 2012 Oct;
7(9):e45663.
PMID: 23029168
Aims: Myocardial fibrosis contributes to the development of heart failure. Activated Protein C (aPC) is a circulating anticoagulant with anti-inflammatory and cytoprotective properties. Using a model of myocardial fibrosis second...
5.
Sopel M, Rosin N, Lee T, Legare J
Lab Invest
. 2010 Dec;
91(4):565-78.
PMID: 21116240
Myocardial fibrosis is characterized by significant extracellular matrix (ECM) deposition. The specific cellular mediators that contribute to the development of fibrosis are not well understood. Using a model of fibrosis...