Miklos Sahin-Toth
Overview
Explore the profile of Miklos Sahin-Toth including associated specialties, affiliations and a list of published articles.
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Articles
152
Citations
3110
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Recent Articles
1.
Sandor M, Nemeth B, Demcsak A, Sahin-Toth M
JCI Insight
. 2025 Mar;
PMID: 40067384
No abstract available.
2.
Sandor M, Vitale D, Nagy Z, Ibrahim S, Abu-El-Haija M, Lazou M, et al.
Pancreatology
. 2024 Dec;
25(1):70-81.
PMID: 39734120
Background/objectives: Genetic variants in PRSS1 encoding human cationic trypsinogen are associated with hereditary pancreatitis. The clinically frequent variants exert their pathogenic effect by increasing intrapancreatic trypsin activity, while a distinct...
3.
Berke G, Sandor M, Xiao X, Lowe M, Ewers M, Eross B, et al.
Sci Rep
. 2024 Dec;
14(1):30965.
PMID: 39730750
The CEL-HYB1 hybrid allele of the carboxyl ester lipase (CEL) gene and its pseudogene (CELP) has been associated with chronic pancreatitis (CP). Recent work indicated that amino acid positions 488...
4.
Nemeth B, Kiss B, Sahin-Toth M, Magyar C, Pal G
Proteins
. 2024 Sep;
93(2):543-554.
PMID: 39301701
Eglin C, a small protein from the medicinal leech, has been long considered a general high-affinity inhibitor of chymotrypsins and elastases. Here, we demonstrate that eglin C inhibits human chymotrypsin-like...
5.
Sandor M, Scheers I, Masamune A, Witt H, LaRusch J, Chen J, et al.
Gut
. 2024 Sep;
74(4):678-679.
PMID: 39256032
No abstract available.
6.
Demcsak A, Shariatzadeh S, Sahin-Toth M
Am J Physiol Gastrointest Liver Physiol
. 2024 Jul;
327(3):G333-G344.
PMID: 38981616
The serine protease chymotrypsin protects the pancreas against pancreatitis by degrading trypsinogen, the precursor to the digestive protease trypsin. Taking advantage of previously generated mouse models with either the gene...
7.
Stefanovics R, Sandor M, Demcsak A, Berke G, Nemeth B, Zhang W, et al.
Pancreatology
. 2024 Jun;
24(5):690-697.
PMID: 38876922
Background: Chymotrypsin C (CTRC) protects the pancreas against unwanted intrapancreatic trypsin activity through degradation of trypsinogen. Loss-of-function CTRC variants increase the risk for chronic pancreatitis (CP). The aim of the...
8.
Demcsak A, Sahin-Toth M
Cell Mol Gastroenterol Hepatol
. 2024 May;
18(3):101361.
PMID: 38768901
Background & Aims: Heterozygous SPINK1 mutations are strong risk factors for chronic pancreatitis in humans, yet heterozygous disruption of mouse Spink1 yielded no pancreatic phenotype. To resolve this contradiction, we...
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