Michail Shipitsin
Overview
Explore the profile of Michail Shipitsin including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
17
Citations
8413
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
11.
Qiu W, Hu M, Sridhar A, Opeskin K, Fox S, Shipitsin M, et al.
Nat Genet
. 2008 Apr;
40(5):650-5.
PMID: 18408720
There is increasing evidence showing that the stromal cells surrounding cancer epithelial cells, rather than being passive bystanders, might have a role in modifying tumor outgrowth. The molecular basis of...
12.
Shipitsin M, Polyak K
Lab Invest
. 2008 Apr;
88(5):459-63.
PMID: 18379567
Cancer is a disease of genes. Inherited or somatic alterations in genes are what make a normal cell ignore growth-controlling signals and form a tumor that eventually leads to the...
13.
Wood L, Parsons D, Jones S, Lin J, Sjoblom T, Leary R, et al.
Science
. 2007 Oct;
318(5853):1108-13.
PMID: 17932254
Human cancer is caused by the accumulation of mutations in oncogenes and tumor suppressor genes. To catalog the genetic changes that occur during tumorigenesis, we isolated DNA from 11 breast...
14.
Shipitsin M, Campbell L, Argani P, Weremowicz S, Bloushtain-Qimron N, Yao J, et al.
Cancer Cell
. 2007 Mar;
11(3):259-73.
PMID: 17349583
Cells with distinct phenotypes including stem-cell-like properties have been proposed to exist in normal human mammary epithelium and breast carcinomas, but their detailed molecular characteristics and clinical significance are unclear....
15.
Lim K, Baines A, Fiordalisi J, Shipitsin M, Feig L, Cox A, et al.
Cancer Cell
. 2005 Jun;
7(6):533-45.
PMID: 15950903
RalGEFs were recently shown to be critical for Ras-mediated transformed and tumorigenic growth of human cells. We now show that the oncogenic activity of these proteins is propagated by activation...
16.
Shipitsin M, Feig L
Mol Cell Biol
. 2004 Jun;
24(13):5746-56.
PMID: 15199131
RalA and RalB constitute a family of highly similar (85% identity) Ras-related GTPases. Recently, active forms of both RalA and RalB have been shown to bind to the exocyst complex,...
17.
Polzin A, Shipitsin M, Goi T, Feig L, Turner T
Mol Cell Biol
. 2002 Feb;
22(6):1714-22.
PMID: 11865051
The Ral proteins are members of the Ras superfamily of GTPases. Because they reside in synaptic vesicles, we used transgenic mice expressing a dominant inhibitory form of Ral to investigate...