Michael T Crow
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Explore the profile of Michael T Crow including associated specialties, affiliations and a list of published articles.
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1867
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Recent Articles
11.
Wu L, Nam Y, Kung G, Crow M, Kitsis R
J Biol Chem
. 2010 Apr;
285(25):19235-45.
PMID: 20392691
Inhibition of apoptosis is critical for carcinogenesis. ARC (apoptosis repressor with caspase recruitment domain) is an endogenous inhibitor of apoptosis that antagonizes both intrinsic and extrinsic apoptosis pathways. Although normally...
12.
Gandolfo M, Jang H, Bagnasco S, Ko G, Agreda P, Satpute S, et al.
Kidney Int
. 2009 Jul;
76(7):717-29.
PMID: 19625990
T lymphocytes modulate early ischemia-reperfusion injury in the kidney; however, their role during repair is unknown. We studied the role of TCRbeta(+)CD4(+)CD25(+)Foxp3(+) regulatory T cells (Tregs), known to blunt immune...
13.
Pulmonary epithelial neuropilin-1 deletion enhances development of cigarette smoke-induced emphysema
Le A, Zielinski R, He C, Crow M, Biswal S, Tuder R, et al.
Am J Respir Crit Care Med
. 2009 Jun;
180(5):396-406.
PMID: 19520907
Rationale: Cigarette smoke (CS) exposure is an important risk factor for chronic obstructive pulmonary disease; however, not all smokers develop disease, suggesting that other factors influence disease development. Objectives: We...
14.
Liu M, Grigoryev D, Crow M, Haas M, Yamamoto M, Reddy S, et al.
Kidney Int
. 2009 May;
76(3):277-85.
PMID: 19436334
Oxidative stress is involved in acute kidney injury due to ischemia-reperfusion and chemotherapy-induced nephrotoxicity. To investigate their basic mechanisms we studied the role of nuclear factor-erythroid 2-p45-related factor 2 (Nrf2),...
15.
Angelini D, Su Q, Yamaji-Kegan K, Fan C, Skinner J, Champion H, et al.
Am J Physiol Lung Cell Mol Physiol
. 2009 Jan;
296(4):L582-93.
PMID: 19136574
Pulmonary hypertension (PH) is a serious disease of multiple etiologies mediated by hypoxia, immune stimuli, and elevated pulmonary pressure that leads to vascular thickening and eventual right heart failure. In...
16.
Szczepaniak W, Zhang Y, Hagerty S, Crow M, Kesari P, Garcia J, et al.
Transl Res
. 2008 Nov;
152(5):213-24.
PMID: 19010292
S1P has been demonstrated to protect against the formation of lipopolysaccharide (LPS)-induced lung edema when administered concomitantly with LPS. In the current study, we sought to determine the effectiveness of...
17.
Zhang M, Koitabashi N, Nagayama T, Rambaran R, Feng N, Takimoto E, et al.
Cell Signal
. 2008 Sep;
20(12):2231-6.
PMID: 18790048
Cyclic GMP-selective phosphodiesterase type 5 (PDE5) has been traditionally thought to play a little role in cardiac myocytes, yet recent studies using selective inhibitors such as sildenafil suggest it can...
18.
Le A, Damico R, Damarla M, Boueiz A, Pae H, Skirball J, et al.
J Appl Physiol (1985)
. 2008 Aug;
105(4):1282-90.
PMID: 18669934
Signaling via p38 MAP kinase has been implicated in the mechanotransduction associated with mechanical stress and ventilator-induced lung injury (VILI). However, the critical downstream mediators of alveolar injury remain incompletely...
19.
Macrophage migration inhibitory factor governs endothelial cell sensitivity to LPS-induced apoptosis
Damico R, Chesley A, Johnston L, Bind E, Amaro E, Nijmeh J, et al.
Am J Respir Cell Mol Biol
. 2008 Feb;
39(1):77-85.
PMID: 18239193
Human endothelial cells (EC) are typically resistant to the apoptotic effects of stimuli associated with lung disease. The determinants of this resistance remain incompletely understood. Macrophage migration inhibitory factor (MIF)...
20.
Li J, Wei H, Chesley A, Moon C, Krawczyk M, Volkova M, et al.
J Biol Chem
. 2007 Oct;
282(48):34984-93.
PMID: 17925408
Pleiotrophin is a development-regulated cytokine and growth factor that can promote angiogenesis, cell proliferation, or differentiation, and it has been reported to have neovasculogenic effects in damaged heart. Developmentally, it...