Mette Hollensted
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Explore the profile of Mette Hollensted including associated specialties, affiliations and a list of published articles.
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30
Citations
1501
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Recent Articles
1.
Gjesing A, Engelbrechtsen L, Thuesen A, Have C, Hollensted M, Grarup N, et al.
Diabetes Res Clin Pract
. 2022 Nov;
194:110159.
PMID: 36400171
Aims: Rare variants in the glucokinase gene (GCK) cause Maturity-Onset Diabetes of the Young (MODY2/GCK-MODY). We investigated the prevalence of GCK variants, phenotypic characteristics, micro- and macrovascular disease at baseline...
2.
Vogelezang S, Bradfield J, Ahluwalia T, Curtin J, Lakka T, Grarup N, et al.
PLoS Genet
. 2020 Oct;
16(10):e1008718.
PMID: 33045005
The genetic background of childhood body mass index (BMI), and the extent to which the well-known associations of childhood BMI with adult diseases are explained by shared genetic factors, are...
3.
Trier C, Hollensted M, Schnurr T, Lund M, Nielsen T, Rui G, et al.
Int J Obes (Lond)
. 2020 Sep;
45(1):66-76.
PMID: 32921795
Objectives: To determine the prevalence of Melanocortin-4 Receptor (MC4R) mutations in a cohort of children and adolescents with overweight or obesity and to determine whether treatment responses differed between carriers...
4.
Bradfield J, Vogelezang S, Felix J, Chesi A, Helgeland O, Horikoshi M, et al.
Hum Mol Genet
. 2019 Sep;
28(19):3327-3338.
PMID: 31504550
Although hundreds of genome-wide association studies-implicated loci have been reported for adult obesity-related traits, less is known about the genetics specific for early-onset obesity and with only a few studies...
5.
Viitasalo A, Schnurr T, Pitkanen N, Hollensted M, Nielsen T, Pahkala K, et al.
Am J Clin Nutr
. 2019 Sep;
110(5):1079-1087.
PMID: 31504107
Background: Mendelian randomization studies in adults suggest that abdominal adiposity is causally associated with increased risk of type 2 diabetes and coronary artery disease in adults, but its causal effect...
6.
Niazi R, Gjesing A, Hollensted M, Have C, Borisevich D, Grarup N, et al.
BMC Med Genet
. 2019 Sep;
20(1):152.
PMID: 31488071
Background: Consanguine families display a high degree of homozygosity which increases the risk of family members suffering from autosomal recessive disorders. Thus, homozygous mutations in monogenic obesity genes may be...
7.
Iepsen E, Zhang J, Hollensted M, Madsbad S, Hansen T, Holst J, et al.
J Bone Miner Metab
. 2019 Sep;
38(1):117-125.
PMID: 31471646
Pathogenic mutations in the melanocortin-4 receptor (MC4R) are associated with obesity, increased linear growth, and higher bone mass in children, and rodent studies have indicated an effect of the MC4R...
8.
Sun J, Have C, Hollensted M, Grarup N, Linneberg A, Pedersen O, et al.
PLoS One
. 2019 Aug;
14(8):e0220805.
PMID: 31415576
Background: Based on the association of common GLIS3 variants with various forms of diabetes and the biological role of GLIS3 in beta-cells, we sequenced GLIS3 in non-diabetic and diabetic Danes...
9.
Genetic predisposition to higher body fat yet lower cardiometabolic risk in children and adolescents
Viitasalo A, Schnurr T, Pitkanen N, Hollensted M, Nielsen T, Pahkala K, et al.
Int J Obes (Lond)
. 2019 Jul;
43(10):2007-2016.
PMID: 31332278
Background: Most obese children show cardiometabolic impairments, such as insulin resistance, dyslipidemia, and hypertension. Yet some obese children retain a normal cardiometabolic profile. The mechanisms underlying this variability remain largely...
10.
Aasbrenn M, Schnurr T, Have C, Svendstrup M, Hansen D, Worm D, et al.
Obes Surg
. 2019 Apr;
29(8):2554-2561.
PMID: 31001758
Background: The weight loss after bariatric surgery shows considerable individual variation. Twin studies of response to dietary interventions and studies of bariatric surgery patients suggest that genetic differences may play...