Matthew E C Bourkas
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Explore the profile of Matthew E C Bourkas including associated specialties, affiliations and a list of published articles.
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9
Citations
119
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Recent Articles
1.
Walsh D, Rees J, Mehra S, Bourkas M, Kaczmarczyk L, Stuart E, et al.
PLoS Pathog
. 2024 Apr;
20(4):e1012087.
PMID: 38557815
Prion diseases uniquely manifest in three distinct forms: inherited, sporadic, and infectious. Wild-type prions are responsible for the sporadic and infectious versions, while mutant prions cause inherited variants like fatal...
2.
Walsh D, Rees J, Mehra S, Bourkas M, Kaczmarczyk L, Stuart E, et al.
bioRxiv
. 2023 Oct;
PMID: 37808761
Prion diseases uniquely manifest in three distinct forms: inherited, sporadic, and infectious. Wild-type prions are responsible for the sporadic and infectious versions, while mutant prions cause inherited variants like fatal...
3.
Bourkas A, Barone N, Bourkas M, Mannarino M, Fraser R, Lorincz A, et al.
BMJ Open
. 2023 Aug;
13(8):e068207.
PMID: 37567745
Objectives: To compare teledermatology and face-to-face (F2F) agreement in primary diagnoses of dermatological conditions. Design: Systematic review and meta-analysis METHODS: MEDLINE, Embase, Cochrane Library (Wiley), CINAHL and medRxiv were searched...
4.
Arshad H, Patel Z, Mehrabian M, Bourkas M, Al-Azzawi Z, Schmitt-Ulms G, et al.
J Biol Chem
. 2021 Aug;
297(3):101073.
PMID: 34390689
The study of prions and the discovery of candidate therapeutics for prion disease have been facilitated by the ability of prions to replicate in cultured cells. Paradigms in which prion...
5.
Arshad H, Bourkas M, Watts J
Prog Mol Biol Transl Sci
. 2020 Sep;
175:179-211.
PMID: 32958232
The transmission of prions between species is typically an inefficient process due to the species barrier, which represents incompatibility between prion seed and substrate molecules. Bank voles (Myodes glareolus) are...
6.
Bourkas M, Arshad H, Al-Azzawi Z, Halgas O, Shikiya R, Mehrabian M, et al.
J Biol Chem
. 2019 Feb;
294(13):4911-4923.
PMID: 30705093
Prions are infectious protein aggregates that cause several fatal neurodegenerative diseases. Prion research has been hindered by a lack of cellular paradigms for studying the replication of prions from different...
7.
Ghodrati F, Mehrabian M, Williams D, Halgas O, Bourkas M, Watts J, et al.
Sci Rep
. 2018 Jun;
8(1):8654.
PMID: 29872131
At times, it can be difficult to discern if a lack of overlap in reported interactions for a protein-of-interest reflects differences in methodology or biology. In such instances, systematic analyses...
8.
Watts J, Bourkas M, Arshad H
Acta Neuropathol
. 2017 Nov;
135(2):159-178.
PMID: 29151170
The essential role of the cellular prion protein (PrP) in prion disorders such as Creutzfeldt-Jakob disease is well documented. Moreover, evidence is accumulating that PrP may act as a receptor...
9.
Watts J, Giles K, Bourkas M, Patel S, Oehler A, Gavidia M, et al.
Acta Neuropathol
. 2016 Jun;
132(4):593-610.
PMID: 27350609
Attempts to model inherited human prion disorders such as familial Creutzfeldt-Jakob disease (CJD), Gerstmann-Sträussler-Scheinker (GSS) disease, and fatal familial insomnia (FFI) using genetically modified mice have produced disappointing results. We...