Matthew DeBerge
Overview
Explore the profile of Matthew DeBerge including associated specialties, affiliations and a list of published articles.
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26
Citations
725
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Recent Articles
1.
Filipp M, Ge Z, DeBerge M, Lantz C, Glinton K, Gao P, et al.
Circulation
. 2025 Mar;
PMID: 40071347
Background: Despite the high morbidity and mortality of heart failure with preserved ejection fraction (HFpEF), treatment options remain limited. The HFpEF syndrome is associated with a high comorbidity burden, including...
2.
Lantz C, Becker A, DeBerge M, Filipp M, Glinton K, Ananthakrishnan A, et al.
Immunity
. 2025 Feb;
58(2):344-361.e7.
PMID: 39938482
In response to organ injury in adults, macrophages often promote scarring, yet during early life, they are required for tissue regeneration. To elucidate the mechanisms underlying age-associated regeneration, we compared...
3.
DeBerge M, Glinton K, Lantz C, Ge Z, Sullivan D, Patil S, et al.
Nat Cardiovasc Res
. 2025 Jan;
4(1):83-101.
PMID: 39747455
Myocardial infarction (MI) mobilizes macrophages, the central protagonists of tissue repair in the infarcted heart. Although necessary for repair, macrophages also contribute to adverse remodeling and progression to heart failure....
4.
DeBerge M, Schroth S, Du F, Yeap X, Wang J, Zhang Z, et al.
Proc Natl Acad Sci U S A
. 2024 Jun;
121(26):e2319623121.
PMID: 38889142
Solid organ transplantation mobilizes myeloid cells, including monocytes and macrophages, which are central protagonists of allograft rejection. However, myeloid cells can also be functionally reprogrammed by perioperative costimulatory blockade to...
5.
Thorp E, Filipp M, Dima M, Tan C, Feinstein M, Popko B, et al.
Brain Behav Immun
. 2024 May;
119:818-835.
PMID: 38735403
Survivors of myocardial infarction are at increased risk for vascular dementia. Neuroinflammation has been implicated in the pathogenesis of vascular dementia, yet little is known about the cellular and molecular...
6.
Ruan W, Li T, Lee J, Bang I, Deng W, Ma X, et al.
Res Sq
. 2024 Mar;
PMID: 38464103
Acute myocardial infarction stands as a prominent cause of morbidity and mortality worldwide. Clinical studies have demonstrated that the severity of cardiac injury following myocardial infarction exhibits a circadian pattern,...
7.
DeBerge M, Chaudhary R, Schroth S, Thorp E
JACC Basic Transl Sci
. 2023 Aug;
8(7):884-904.
PMID: 37547069
Immune cell function among the myocardium, now more than ever, is appreciated to regulate cardiac function and pathophysiology. This is the case for both innate immunity, which includes neutrophils, monocytes,...
8.
Slysz J, Sinha A, DeBerge M, Singh S, Avgousti H, Lee I, et al.
JCI Insight
. 2023 Jul;
8(17).
PMID: 37471165
Femoral atherosclerotic plaques are less inflammatory than carotid plaques histologically, but limited cell-level data exist regarding comparative immune landscapes and polarization at these sites. We investigated intraplaque leukocyte phenotypes and...
9.
Glinton K, Ma W, Lantz C, Grigoryeva L, DeBerge M, Liu X, et al.
J Clin Invest
. 2022 Mar;
132(9).
PMID: 35271504
Clearance of dying cells by efferocytosis is necessary for cardiac repair after myocardial infarction (MI). Recent reports have suggested a protective role for vascular endothelial growth factor C (VEGFC) during...
10.
Thorp E, Flanagan M, Popko B, DeBerge M
Semin Immunol
. 2022 Mar;
59:101600.
PMID: 35227567
Myocardial infarction is associated with increased risk for vascular dementia. In both myocardial infarction and vascular dementia, there is evidence that elevated inflammatory biomarkers are associated with worsened clinical outcomes....