Mark K Nohr
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Explore the profile of Mark K Nohr including associated specialties, affiliations and a list of published articles.
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10
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811
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Recent Articles
1.
von Voss L, Arora T, Assis J, Kuentzel K, Arfelt K, Nohr M, et al.
J Endocr Soc
. 2024 Nov;
8(12):bvae188.
PMID: 39545055
Context: Excessive eating and intake of a Western diet negatively affect the intestinal immune system, resulting in compromised glucose homeostasis and lower gut bacterial diversity. The G protein-coupled receptor GPR183...
2.
Luck H, Khan S, Kim J, Copeland J, Revelo X, Tsai S, et al.
Nat Commun
. 2019 Aug;
10(1):3650.
PMID: 31409776
The intestinal immune system is emerging as an important contributor to obesity-related insulin resistance, but the role of intestinal B cells in this context is unclear. Here, we show that...
3.
Ghazarian M, Revelo X, Nohr M, Luck H, Zeng K, Lei H, et al.
Sci Immunol
. 2017 Jun;
2(10).
PMID: 28567448
Obesity-related insulin resistance is driven by low-grade chronic inflammation of metabolic tissues. In the liver, non-alcoholic fatty liver disease (NAFLD) is associated with hepatic insulin resistance and systemic glucose dysregulation....
4.
Nohr M, Bobba N, Richelsen B, Lund S, Pedersen S
Int J Mol Sci
. 2017 May;
18(5).
PMID: 28481291
Brown adipose tissue thermogenesis at the cost of energy is not only important for the development of obesity, but also possesses great promise in anti-obesity treatment. Uncoupling protein 1 (UCP1)...
5.
Nohr M, Kroager T, Sanggaard K, Knudsen A, Stensballe A, Enghild J, et al.
PLoS One
. 2016 Jul;
11(7):e0159747.
PMID: 27438462
Adipose tissue inflammation is believed to play a pivotal role in the development obesity-related morbidities such as insulin resistance. However, it is not known how this (low-grade) inflammatory state develops....
6.
Nohr M, Dudele A, Poulsen M, Ebbesen L, Radko Y, Christensen L, et al.
PLoS One
. 2016 Jan;
11(1):e0146840.
PMID: 26751381
Unlabelled: Low-grade inflammation is seen with obesity and is suggested to be a mediator of insulin resistance. The eliciting factor of low-grade inflammation is unknown but increased permeability of gut...
7.
Grunddal K, Ratner C, Svendsen B, Sommer F, Engelstoft M, Madsen A, et al.
Endocrinology
. 2015 Oct;
157(1):176-94.
PMID: 26469136
The 2 gut hormones glucagon-like peptide-1 (GLP-1) and peptide YY (PYY) are well known to be coexpressed, costored, and released together to coact in the control of key metabolic target...
8.
Engelstoft M, Park W, Sakata I, Kristensen L, Husted A, Osborne-Lawrence S, et al.
Mol Metab
. 2013 Dec;
2(4):376-92.
PMID: 24327954
The molecular mechanisms regulating secretion of the orexigenic-glucoregulatory hormone ghrelin remain unclear. Based on qPCR analysis of FACS-purified gastric ghrelin cells, highly expressed and enriched 7TM receptors were comprehensively identified...
9.
Nohr M, Pedersen M, Gille A, Egerod K, Engelstoft M, Husted A, et al.
Endocrinology
. 2013 Jul;
154(10):3552-64.
PMID: 23885020
The expression of short-chain fatty acid receptors GPR41/FFAR3 and GPR43/ free fatty acid receptor 2 (FFAR2) was studied in the gastrointestinal tract of transgenic monomeric red fluorescent protein (mRFP) reporter...
10.
Egerod K, Engelstoft M, Grunddal K, Nohr M, Secher A, Sakata I, et al.
Endocrinology
. 2012 Oct;
153(12):5782-95.
PMID: 23064014
Enteroendocrine cells such as duodenal cholecystokinin (CCK cells) are generally thought to be confined to certain segments of the gastrointestinal (GI) tract and to store and release peptides derived from...