Maksymilian Prondzynski
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Explore the profile of Maksymilian Prondzynski including associated specialties, affiliations and a list of published articles.
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Citations
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Recent Articles
11.
Sweat M, Cao Y, Zhang X, Burnicka-Turek O, Perez-Cervantes C, Akerberg B, et al.
bioRxiv
. 2023 May;
PMID: 37131696
Understanding how the atrial and ventricular chambers of the heart maintain their distinct identity is a prerequisite for treating chamber-specific diseases. Here, we selectively inactivated the transcription factor in the...
12.
Cao Y, Zhang X, Akerberg B, Yuan H, Sakamoto T, Xiao F, et al.
Circulation
. 2023 Jan;
147(11):881-896.
PMID: 36705030
Background: Cardiac chamber-selective transcriptional programs underpin the structural and functional differences between atrial and ventricular cardiomyocytes (aCMs and vCMs). The mechanisms responsible for these chamber-selective transcriptional programs remain largely undefined....
13.
Kumar R, Mancebo J, Patenaude R, Sack K, Prondzynski M, Packard A, et al.
Adv Mater
. 2022 Sep;
34(47):e2207376.
PMID: 36153826
Colloids, known as volume expanders, have been used as resuscitation fluids for hypovolemic shock for decades, as they increase plasma oncotic pressure and expand intravascular volume. However, recent studies show...
14.
Zech A, Prondzynski M, Singh S, Pietsch N, Orthey E, Alizoti E, et al.
Cells
. 2022 Sep;
11(17).
PMID: 36078153
Genetic variants in α-actinin-2 (ACTN2) are associated with several forms of (cardio)myopathy. We previously reported a heterozygous missense (c.740C>T) gene variant, associated with hypertrophic cardiomyopathy, and characterized by an electro-mechanical...
15.
Disease modeling of a mutation in α-actinin 2 guides clinical therapy in hypertrophic cardiomyopathy
Prondzynski M, Lemoine M, Zech A, Horvath A, Di Mauro V, Koivumaki J, et al.
EMBO Mol Med
. 2022 Aug;
14(8):e16423.
PMID: 35938313
No abstract available.
16.
Liu X, Wang S, Guo X, Li Y, Ogurlu R, Lu F, et al.
Circulation
. 2021 Apr;
143(19):1894-1911.
PMID: 33793303
Background: Mutations in tafazzin (), a gene required for biogenesis of cardiolipin, the signature phospholipid of the inner mitochondrial membrane, causes Barth syndrome (BTHS). Cardiomyopathy and risk of sudden cardiac...
17.
Bezzerides V, Prondzynski M, Carrier L, Pu W
Cardiovasc Res
. 2020 Apr;
116(9):1635-1650.
PMID: 32321160
Inherited arrhythmias are disorders caused by one or more genetic mutations that increase the risk of arrhythmia, which result in life-long risk of sudden death. These mutations either primarily perturb...
18.
Horvath A, Christ T, Koivumaki J, Prondzynski M, Zech A, Spohn M, et al.
Cells
. 2020 Jan;
9(1).
PMID: 31968557
Human induced pluripotent stem cell-derived cardiomyocytes (hiPSC-CMs) represent an unlimited source of human CMs that could be a standard tool in drug research. However, there is concern whether hiPSC-CMs express...
19.
Disease modeling of a mutation in α-actinin 2 guides clinical therapy in hypertrophic cardiomyopathy
Prondzynski M, Lemoine M, Zech A, Horvath A, Di Mauro V, Koivumaki J, et al.
EMBO Mol Med
. 2019 Nov;
11(12):e11115.
PMID: 31680489
Hypertrophic cardiomyopathy (HCM) is a cardiac genetic disease accompanied by structural and contractile alterations. We identified a rare c.740C>T (p.T247M) mutation in ACTN2, encoding α-actinin 2 in a HCM patient,...
20.
Lemme M, Braren I, Prondzynski M, Aksehirlioglu B, Ulmer B, Schulze M, et al.
Cardiovasc Res
. 2019 Oct;
116(8):1487-1499.
PMID: 31598634
Aims: Chronic tachypacing is commonly used in animals to induce cardiac dysfunction and to study mechanisms of heart failure and arrhythmogenesis. Human induced pluripotent stem cells (hiPSC) may replace animal...