Makiyeh Tohidast-Akrad
Overview
Explore the profile of Makiyeh Tohidast-Akrad including associated specialties, affiliations and a list of published articles.
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11
Citations
514
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Recent Articles
1.
Becker T, Tohidast-Akrad M, Humpeler S, Gerlag D, Kiener H, Zenz P, et al.
Acta Histochem
. 2014 Aug;
116(7):1199-207.
PMID: 25109449
Patients with rheumatoid arthritis (RA) show modulated circadian rhythms of inflammatory cytokines and cortisol, which may be associated with a modified expression of clock genes. The expression of major clock...
2.
Jimenez-Boj E, Kedersha N, Tohidast-Akrad M, Karlhofer F, Stummvoll G, Zimmermann C, et al.
Arthritis Rheum
. 2008 Apr;
58(5):1226-36.
PMID: 18438838
Objective: T cell intracytoplasmic antigen 1 (TIA-1) and TIA-1-related protein (TIAR) are involved in posttranscriptional regulation of the expression of tumor necrosis factor alpha (TNFalpha) and other proteins. Given the...
3.
Bobacz K, Sunk I, Hayer S, Amoyo L, Tohidast-Akrad M, Kollias G, et al.
Arthritis Rheum
. 2008 Jan;
58(1):109-18.
PMID: 18163510
Objective: To examine whether the endogenous expression of growth differentiation factor 5 (GDF-5) and bone morphogenetic protein 7 (BMP-7) is altered in the cartilage and synovium of human tumor necrosis...
4.
Hoffmann M, Tuncel J, Skriner K, Tohidast-Akrad M, Turk B, Pinol-Roma S, et al.
J Immunol
. 2007 Nov;
179(11):7568-76.
PMID: 18025202
A single intradermal injection of the mineral oil pristane in susceptible DA.1F rats induces erosive arthritis closely mimicking rheumatoid arthritis (RA). Pristane-induced arthritis (PIA) is driven by autoreactive T cells...
5.
Korb A, Tohidast-Akrad M, Cetin E, Axmann R, Smolen J, Schett G
Arthritis Rheum
. 2006 Sep;
54(9):2745-56.
PMID: 16947383
Objective: Activation of p38 MAPK is a key signaling step in chronic inflammation. Inhibition of p38 MAPK is considered to be a promising future strategy to control inflammatory diseases, but...
6.
Hayer S, Tohidast-Akrad M, Haralambous S, Jahn-Schmid B, Skriner K, Trembleau S, et al.
J Immunol
. 2005 Dec;
175(12):8327-36.
PMID: 16339574
Human TNF-alpha transgenic (hTNFtg) mice develop erosive arthritis closely resembling rheumatoid arthritis (RA). To investigate mechanisms leading to pathological autoimmune reactions in RA, we examined hTNFtg animals for the presence...
7.
Hayer S, Steiner G, Gortz B, Reiter E, Tohidast-Akrad M, Amling M, et al.
J Exp Med
. 2005 Mar;
201(6):903-14.
PMID: 15781582
Chronic inflammation is a major trigger of local and systemic bone loss. Disintegration of cell-matrix interaction is a prerequisite for the invasion of inflammatory tissue into bone. CD44 is a...
8.
Zwerina J, Hayer S, Tohidast-Akrad M, Bergmeister H, Redlich K, Feige U, et al.
Arthritis Rheum
. 2004 Jan;
50(1):277-90.
PMID: 14730626
Objective: To investigate the efficacy of single and combined blockade of tumor necrosis factor (TNF), interleukin-1 (IL-1), and RANKL pathways on synovial inflammation, bone erosion, and cartilage destruction in a...
9.
Redlich K, Hayer S, Ricci R, David J, Tohidast-Akrad M, Kollias G, et al.
J Clin Invest
. 2002 Nov;
110(10):1419-27.
PMID: 12438440
The detailed cellular and molecular mechanisms leading to joint destruction in rheumatoid arthritis, a disease driven by proinflammatory cytokines, are still unknown. To address the question of whether osteoclasts play...
10.
Fritsch R, Eselbock D, Skriner K, Jahn-Schmid B, Scheinecker C, Bohle B, et al.
J Immunol
. 2002 Jul;
169(2):1068-76.
PMID: 12097415
The role of autoimmune reactions in the pathogenesis of rheumatoid arthritis (RA) is poorly understood. To address this issue we have investigated the spontaneous T cell response to two well-characterized...