Maia Parsadanian
Overview
Explore the profile of Maia Parsadanian including associated specialties, affiliations and a list of published articles.
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Articles
27
Citations
3098
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0
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Recent Articles
1.
Wu W, Hill S, Nathan W, Paiano J, Callen E, Wang D, et al.
Nature
. 2021 Mar;
593(7859):440-444.
PMID: 33767446
Defects in DNA repair frequently lead to neurodevelopmental and neurodegenerative diseases, underscoring the particular importance of DNA repair in long-lived post-mitotic neurons. The cellular genome is subjected to a constant...
2.
Main B, Villapol S, Sloley S, Barton D, Parsadanian M, Agbaegbu C, et al.
Mol Neurodegener
. 2018 Apr;
13(1):17.
PMID: 29618365
Background: Traumatic Brain Injury (TBI) is a major cause of disability and mortality, to which there is currently no comprehensive treatment. Blood Brain Barrier (BBB) dysfunction is well documented in...
3.
Neustadtl A, Winston C, Parsadanian M, Main B, Villapol S, Burns M
Neuroreport
. 2017 May;
28(10):618-624.
PMID: 28542068
Synaptic loss is a symptom of Alzheimer's disease (AD) that is associated with the onset of cognitive decline and the loss of executive function. The strongest genetic risk factor for...
4.
Winston C, Noel A, Neustadtl A, Parsadanian M, Barton D, Chellappa D, et al.
Am J Pathol
. 2016 Feb;
186(3):552-67.
PMID: 26857506
Mild traumatic brain injury (mTBI) is an emerging risk for chronic behavioral, cognitive, and neurodegenerative conditions. Athletes absorb several hundred mTBIs each year; however, rodent models of repeat mTBI (rmTBI)...
5.
Washington P, Morffy N, Parsadanian M, Zapple D, Burns M
J Neurotrauma
. 2013 Sep;
31(1):125-34.
PMID: 24050316
Soluble amyloid-beta (Aβ) oligomers are hypothesized to be the pathogenic species in Alzheimer's disease (AD), and increased levels of oligomers in the brain subsequent to traumatic brain injury (TBI) may...
6.
Bachis A, Avdoshina V, Zecca L, Parsadanian M, Mocchetti I
J Neurosci
. 2012 Jul;
32(28):9477-84.
PMID: 22787033
The molecular mechanisms leading to synaptic simplification and neuronal apoptosis in human immunodeficiency virus type 1 (HIV-1)-positive subjects are unknown. The HIV protein gp120 reduced the length of neuronal processes...
7.
Wildsmith K, Basak J, Patterson B, Pyatkivskyy Y, Kim J, Yarasheski K, et al.
PLoS One
. 2012 Jun;
7(6):e38013.
PMID: 22675504
Apolipoprotein E (ApoE) is the strongest genetic risk factor for Alzheimer's disease and has been implicated in the risk for other neurological disorders. The three common ApoE isoforms (ApoE2, E3,...
8.
Washington P, Forcelli P, Wilkins T, Zapple D, Parsadanian M, Burns M
J Neurotrauma
. 2012 May;
29(13):2283-96.
PMID: 22642287
Traumatic brain injury (TBI) can cause a broad array of behavioral problems including cognitive and emotional deficits. Human studies comparing neurobehavioral outcomes after TBI suggest that cognitive impairments increase with...
9.
Basak J, Kim J, Pyatkivskyy Y, Wildsmith K, Jiang H, Parsadanian M, et al.
Mol Neurodegener
. 2012 Apr;
7:14.
PMID: 22512932
Background: Abnormal proteostasis due to alterations in protein turnover has been postulated to play a central role in several neurodegenerative diseases. Therefore, the development of techniques to quantify protein turnover...
10.
Avdoshina V, Becker J, Campbell L, Parsadanian M, Mhyre T, Tessarollo L, et al.
J Neurovirol
. 2010 Dec;
17(1):58-62.
PMID: 21165786
In the central nervous system, chemokines are primarily mediators of inflammatory processes. Their receptors, in particular, CXCR4 and CCR5, serve as co-factors along with CD4 that permit Human immunodeficiency virus-1...