Luke Ames
Overview
Explore the profile of Luke Ames including associated specialties, affiliations and a list of published articles.
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Articles
7
Citations
38
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0
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Recent Articles
1.
Coleman D, Keane P, Chin P, Ames L, Kellaway S, Blair H, et al.
iScience
. 2024 Apr;
27(4):109576.
PMID: 38638836
AML is characterized by mutations in genes associated with growth regulation such as internal tandem duplications (ITD) in the receptor kinase FLT3. Inhibitors targeting FLT3 (FLT3i) are being used to...
2.
Kellaway S, Potluri S, Keane P, Blair H, Ames L, Worker A, et al.
Nat Commun
. 2024 Feb;
15(1):1359.
PMID: 38355578
Acute Myeloid Leukemia (AML) is caused by multiple mutations which dysregulate growth and differentiation of myeloid cells. Cells adopt different gene regulatory networks specific to individual mutations, maintaining a rapidly...
3.
Coleman D, Keane P, Luque-Martin R, Chin P, Blair H, Ames L, et al.
Cell Rep
. 2023 Dec;
42(12):113568.
PMID: 38104314
Acute myeloid leukemia (AML) is a heterogeneous disease caused by different mutations. Previously, we showed that each mutational subtype develops its specific gene regulatory network (GRN) with transcription factors interacting...
4.
Coleman D, Keane P, Luque-Martin R, Chin P, Blair H, Ames L, et al.
bioRxiv
. 2023 Jul;
PMID: 37503022
AML is a heterogenous disease caused by different mutations. We have previously shown that each mutational sub-type develops its specific gene regulatory network (GRN) with transcription factors interacting with multiple...
5.
Adamo A, Chin P, Keane P, Assi S, Potluri S, Kellaway S, et al.
Leukemia
. 2022 Nov;
37(1):102-112.
PMID: 36333583
Acute myeloid leukemia (AML) is a heterogeneous hematological malignancy caused by mutations in genes encoding transcriptional and epigenetic regulators together with signaling genes. It is characterized by a disturbance of...
6.
Ptasinska A, Pickin A, Assi S, Chin P, Ames L, Avellino R, et al.
Cell Rep
. 2019 Nov;
29(7):2120.
PMID: 31722222
No abstract available.
7.
Ptasinska A, Pickin A, Assi S, Chin P, Ames L, Avellino R, et al.
Cell Rep
. 2019 Sep;
28(12):3022-3031.e7.
PMID: 31533028
Acute myeloid leukemia (AML) is associated with mutations in transcriptional and epigenetic regulator genes impairing myeloid differentiation. The t(8;21)(q22;q22) translocation generates the RUNX1-ETO fusion protein, which interferes with the hematopoietic...