Lori L Wallrath
Overview
Explore the profile of Lori L Wallrath including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
45
Citations
1340
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Herndon M, Ayers M, Gibson-Corley K, Wendt M, Wallrath L, Henry M, et al.
Dis Model Mech
. 2024 Aug;
17(9).
PMID: 39104192
Epithelial-mesenchymal transitions (EMTs) are thought to promote metastasis via downregulation of E-cadherin (also known as Cdh1) and upregulation of mesenchymal markers such as N-cadherin (Cdh2) and vimentin (Vim). Contrary to...
2.
Mohar N, Cox E, Adelizzi E, Moore S, Mathews K, Darbro B, et al.
Int J Mol Sci
. 2024 May;
25(9).
PMID: 38732148
Mutations in the gene-encoding A-type lamins can cause Limb-Girdle muscular dystrophy Type 1B (LGMD1B). This disease presents with weakness and wasting of the proximal skeletal muscles and has a variable...
3.
Wallace M, Zahr H, Perati S, Morsink C, Johnson L, Gacita A, et al.
Mol Biol Cell
. 2023 Aug;
:mbcE21100527.
PMID: 37585285
The gene encodes the nuclear envelope proteins Lamins A and C, which comprise a major part of the nuclear lamina, provide mechanical support to the nucleus, and participate in diverse...
4.
Walker S, Langland C, Viles J, Hecker L, Wallrath L
Cells
. 2023 May;
12(8).
PMID: 37190051
Mutations in the gene cause a collection of diseases known as laminopathies, including muscular dystrophies, lipodystrophies, and early-onset aging syndromes. The gene encodes A-type lamins, lamins A/C, intermediate filaments that...
5.
Shaw N, Rios-Monterrosa J, Fedorchak G, Ketterer M, Coombs G, Lammerding J, et al.
Front Cell Dev Biol
. 2022 Sep;
10:934586.
PMID: 36120560
The nuclei of multinucleated skeletal muscles experience substantial external force during development and muscle contraction. Protection from such forces is partly provided by lamins, intermediate filaments that form a scaffold...
6.
Wallrath L, Rodriguez-Tirado F, Geyer P
Cells
. 2022 Feb;
11(3).
PMID: 35159140
Heterochromatin has historically been considered the dark side of the genome. In part, this reputation derives from its concentration near centromeres and telomeres, regions of the genome repressive to nuclear...
7.
Coombs G, Rios-Monterrosa J, Lai S, Dai Q, Goll A, Ketterer M, et al.
Redox Biol
. 2021 Dec;
48:102196.
PMID: 34872044
Mutations in the human LMNA gene cause a collection of diseases called laminopathies, which includes muscular dystrophy and dilated cardiomyopathy. The LMNA gene encodes lamins, filamentous proteins that form a...
8.
Hinz B, Walker S, Xiong A, Gogal R, Schnieders M, Wallrath L
Int J Mol Sci
. 2021 Oct;
22(20).
PMID: 34681887
Mutations in the gene cause diseases called laminopathies. encodes lamins A and C, intermediate filaments with multiple roles at the nuclear envelope. mutations are frequently single base changes that cause...
9.
Earle A, Kirby T, Fedorchak G, Isermann P, Patel J, Iruvanti S, et al.
Nat Mater
. 2019 Dec;
19(4):464-473.
PMID: 31844279
Mutations in the LMNA gene, which encodes the nuclear envelope (NE) proteins lamins A/C, cause Emery-Dreifuss muscular dystrophy, congenital muscular dystrophy and other diseases collectively known as laminopathies. The mechanisms...
10.
Chandran S, Suggs J, Wang B, Han A, Bhide S, Cryderman D, et al.
Hum Mol Genet
. 2018 Sep;
28(3):351-371.
PMID: 30239736
Laminopathies are diseases caused by dominant mutations in the human LMNA gene encoding A-type lamins. Lamins are intermediate filaments that line the inner nuclear membrane, provide structural support for the...