Kazutomo Sawai
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Explore the profile of Kazutomo Sawai including associated specialties, affiliations and a list of published articles.
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10
Citations
703
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Recent Articles
1.
Tsuda H, Yamahara K, Ishikane S, Otani K, Nakamura A, Sawai K, et al.
Am J Physiol Renal Physiol
. 2010 Aug;
299(5):F1004-13.
PMID: 20739390
Mesenchymal stem cells (MSC) have been reported to be an attractive therapeutic cell source for the treatment of renal diseases. Recently, we reported that transplantation of allogenic fetal membrane-derived MSC...
2.
Tokudome T, Kishimoto I, Yamahara K, Osaki T, Minamino N, Horio T, et al.
Arterioscler Thromb Vasc Biol
. 2009 Jul;
29(10):1516-21.
PMID: 19628785
Objective: Atrial and brain natriuretic peptides (ANP and BNP, respectively) function via guanylyl cyclase (GC)-A, resulting in diuresis, natriuresis, and blood vessel dilation. Here, we investigated the role of endogenous...
3.
Sawai K, Mukoyama M, Mori K, Kasahara M, Koshikawa M, Yokoi H, et al.
Am J Physiol Renal Physiol
. 2007 Aug;
293(4):F1363-72.
PMID: 17699553
CCN1 (cysteine-rich protein 61; Cyr61) is an extracellular matrix-associated signaling molecule that functions in cell migration, adhesion, and differentiation. We previously reported that CCN1 is induced at podocytes in rat...
4.
Makino H, Miyamoto Y, Sawai K, Mori K, Mukoyama M, Nakao K, et al.
Diabetes
. 2006 Sep;
55(10):2747-56.
PMID: 17003339
Glomerular injury plays a pivotal role in the development of diabetic nephropathy. To elucidate molecular mechanisms underlying diabetic glomerulopathy, we compared glomerular gene expression profiles of db/db mice with those...
5.
Sawai K, Mukoyama M, Mori K, Yokoi H, Koshikawa M, Yoshioka T, et al.
Nephrol Dial Transplant
. 2006 Jul;
21(9):2472-7.
PMID: 16820374
Background: Significance of podocyte injury in the progression of diabetic nephropathy is not well-understood. In this study, we examined whether alteration of gap junction protein connexin43 (Cx43) expression in podocytes...
6.
Koshikawa M, Mukoyama M, Mori K, Suganami T, Sawai K, Yoshioka T, et al.
J Am Soc Nephrol
. 2005 Jul;
16(9):2690-701.
PMID: 15987752
Podocytes play an important role in maintaining normal glomerular function and structure, and podocyte injury leads to proteinuria and glomerulosclerosis. The family of mitogen-activated protein kinases (MAPK; extracellular signal-regulated kinase...
7.
Mori K, Lee H, Rapoport D, Drexler I, Foster K, Yang J, et al.
J Clin Invest
. 2005 Feb;
115(3):610-21.
PMID: 15711640
Neutrophil gelatinase-associated lipocalin (Ngal), also known as siderocalin, forms a complex with iron-binding siderophores (Ngal:siderophore:Fe). This complex converts renal progenitors into epithelial tubules. In this study, we tested the hypothesis...
8.
Suganami T, Mukoyama M, Mori K, Yokoi H, Koshikawa M, Sawai K, et al.
FASEB J
. 2004 Oct;
19(1):127-9.
PMID: 15496495
Diabetic nephropathy is the leading cause of end-stage renal disease, for which effective therapy to prevent the progression at advanced stages remains to be established. There is also a long...
9.
Yokoi H, Mukoyama M, Nagae T, Mori K, Suganami T, Sawai K, et al.
J Am Soc Nephrol
. 2004 May;
15(6):1430-40.
PMID: 15153554
Connective tissue growth factor (CTGF/CCN2) is one of the candidate factors mediating fibrogenic activity of TGF-beta. It was shown previously that the blockade of CTGF by antisense oligonucleotide (ODN) inhibits...
10.
Sawai K, Mori K, Mukoyama M, Sugawara A, Suganami T, Koshikawa M, et al.
J Am Soc Nephrol
. 2003 Apr;
14(5):1154-63.
PMID: 12707386
Dynamic recovery of glomerular structure occurs after severe glomerular damage in anti-Thy-1 glomerulonephritis (Thy-1 GN), but its mechanism remains to be investigated. To identify candidate genes possibly involved in glomerular...