Jimmy D Ballard
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Explore the profile of Jimmy D Ballard including associated specialties, affiliations and a list of published articles.
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79
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1658
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Recent Articles
31.
Lanis J, Heinlen L, James J, Ballard J
PLoS Pathog
. 2013 Aug;
9(8):e1003523.
PMID: 23935501
The Clostridium difficile exotoxin, TcdB, which is a major virulence factor, varies between strains of this pathogen. Herein, we show that TcdB from the epidemic BI/NAP1/027 strain of C. difficile...
32.
Toxin inhibition of antimicrobial factors induced by Bacillus anthracis peptidoglycan in human blood
Barua S, Iyer J, Larabee J, Raisley B, Hughes M, Coggeshall K, et al.
Infect Immun
. 2013 Jul;
81(10):3693-702.
PMID: 23876807
Here, we describe the capacity of Bacillus anthracis peptidoglycan (BaPGN) to trigger an antimicrobial response in human white blood cells (WBCs). Analysis of freshly isolated human blood cells found that...
33.
Larabee J, Shakir S, Barua S, Ballard J
J Biol Chem
. 2013 Jun;
288(30):21526-36.
PMID: 23775085
In cells of the innate immune system, pathological increases in intracellular cAMP attenuate immune responses and contribute to infections by bacteria such as Bacillus anthracis. In this work, cAMP from...
34.
Shakir F, Ali T, Bigham A, Ballard J, Miner Jr P, Philpott J
Gastroenterol Hepatol (N Y)
. 2012 Aug;
8(5):313-7.
PMID: 22933861
Clostridium difficile infection has increased in prevalence among patients with inflammatory bowel disease (IBD). Serum antibodies against C. difficile toxins have been detected in susceptible populations and may be protective;...
35.
Lanis J, Hightower L, Shen A, Ballard J
Mol Microbiol
. 2012 Mar;
84(1):66-76.
PMID: 22372854
TcdB, an intracellular bacterial toxin that inactivates small GTPases, is a major Clostridium difficile virulence factor. Recent studies have found that TcdB produced by emerging/hypervirulent strains of C. difficile is...
36.
Rose K, Long P, Shankar M, Ballard J, Webb C
Cell Immunol
. 2011 Nov;
272(2):175-81.
PMID: 22082566
Lethal toxin, a key virulence factor produced by Bacillus anthracis, induces cell death, in part by disrupting numerous signaling pathways, in mouse macrophages. However, exposure to sublethal doses of lethal...
37.
Barua S, Larabee J, Regens J, Ballard J
Environ Sci Pollut Res Int
. 2011 Sep;
19(3):619-27.
PMID: 21881907
Purpose: The aim of this study is to determine whether exposure to hazardous chemicals alters chemokine or cytokine production in macrophages and link these events to changes in intracellular signaling...
38.
Devera T, Joshi S, Aye L, Lang G, Ballard J, Lang M
PLoS One
. 2011 Aug;
6(8):e23817.
PMID: 21858226
Activation of Natural Killer-like T cells (NKT) with the CD1d ligand α-GC leads to enhanced production of anthrax toxin protective Ag (PA)-neutralizing Abs, yet the underlying mechanism for this adjuvant...
39.
Larabee J, Maldonado-Arocho F, Pacheco S, France B, DeGiusti K, Shakir S, et al.
Infect Immun
. 2011 May;
79(8):3302-8.
PMID: 21576335
Anthrax edema toxin (ET) is one of two binary toxins produced by Bacillus anthracis that contributes to the virulence of this pathogen. ET is an adenylate cyclase that generates high...
40.
Larabee J, Shakir S, Hightower L, Ballard J
J Biol Chem
. 2011 Apr;
286(22):19364-72.
PMID: 21487015
The production of cAMP from Bacillus anthracis edema toxin (ET) activates gene expression in macrophages through a complex array of signaling pathways, most of which remain poorly defined. In this...