Jie Hong Hu
Overview
Explore the profile of Jie Hong Hu including associated specialties, affiliations and a list of published articles.
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Articles
17
Citations
473
Followers
0
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Recent Articles
1.
Zhu J, Angelov S, Alp Yildirim I, Wei H, Hu J, Majesky M, et al.
Arterioscler Thromb Vasc Biol
. 2021 Apr;
41(6):1956-1971.
PMID: 33853348
[Figure: see text].
2.
Angelov S, Hu J, Wei H, Airhart N, Shi M, Dichek D
Arterioscler Thromb Vasc Biol
. 2017 Jul;
37(11):2102-2113.
PMID: 28729364
Objective: The role of TGF-β (transforming growth factor-β) signaling in abdominal aortic aneurysm (AAA) formation is controversial. Others reported that systemic blockade of TGF-β by neutralizing antibodies accelerated AAA development...
3.
Angelov S, Zhu J, Hu J, Dichek D
Arterioscler Thromb Vasc Biol
. 2017 Apr;
37(5):740-742.
PMID: 28446468
No abstract available.
4.
Wei H, Hu J, Angelov S, Fox K, Yan J, Enstrom R, et al.
J Am Heart Assoc
. 2017 Jan;
6(1).
PMID: 28119285
Background: Marfan syndrome (MFS) is caused by mutations in the gene encoding fibrillin-1 (FBN1); however, the mechanisms through which fibrillin-1 deficiency causes MFS-associated aortopathy are uncertain. Recently, attention was focused...
5.
Hu J, Wei H, Jaffe M, Airhart N, Du L, Angelov S, et al.
Arterioscler Thromb Vasc Biol
. 2015 Oct;
35(12):2647-56.
PMID: 26494233
Objective: Prenatal deletion of the type II transforming growth factor-β (TGF-β) receptor (TBRII) prevents normal vascular morphogenesis and smooth muscle cell (SMC) differentiation, causing embryonic death. The role of TBRII...
6.
Hu J, Touch P, Zhang J, Wei H, Liu S, Lund I, et al.
Cardiovasc Res
. 2015 Jan;
105(3):372-82.
PMID: 25616415
Aims: Elevated activity of urokinase plasminogen activator (uPA) and MMPs in human arteries is associated with accelerated atherosclerosis, aneurysms, and plaque rupture. We used Apoe-null mice with macrophage-specific uPA overexpression...
7.
Naik V, Leaf E, Hu J, Yang H, Nguyen N, Giachelli C, et al.
Cardiovasc Res
. 2012 Mar;
94(3):545-54.
PMID: 22436847
Aims: Vascular cartilaginous metaplasia and calcification are common in patients with atherosclerosis. However, sources of cells contributing to the development of this complication are currently unknown. In this study, we...
8.
Farris S, Hu J, Krishnan R, Emery I, Chu T, Du L, et al.
J Biol Chem
. 2011 May;
286(25):22665-77.
PMID: 21536666
Data from clinical studies, cell culture, and animal models implicate the urokinase plasminogen activator (uPA)/uPA receptor (uPAR)/plasminogen system in the development of atherosclerosis and aneurysms. However, the mechanisms through which...
9.
Hu J, Du L, Chu T, Otsuka G, Dronadula N, Jaffe M, et al.
Circulation
. 2010 Mar;
121(14):1637-44.
PMID: 20351234
Background: The mechanisms of atherosclerotic plaque rupture are poorly understood. Urokinase-type plasminogen activator (uPA) is expressed at elevated levels by macrophages in advanced human plaques. Patients with evidence of increased...
10.
Krishnan R, Kremen M, Hu J, Emery I, Farris S, Slezicki K, et al.
Arterioscler Thromb Vasc Biol
. 2009 Sep;
29(11):1737-44.
PMID: 19729604
Objective: Enhanced plasminogen activation, mediated by overexpression of urokinase-type plasminogen activator (uPA), accelerates atherosclerosis in apolipoprotein E-null mice. However, the mechanisms through which uPA acts remain unclear. In addition, although...