Jeroen J T Otten
Overview
Explore the profile of Jeroen J T Otten including associated specialties, affiliations and a list of published articles.
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6
Citations
109
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Recent Articles
1.
Medina I, Wieland E, Temmerman L, Otten J, Bermudez B, Bot I, et al.
Eur J Immunol
. 2024 Sep;
54(11):e2350943.
PMID: 39233527
Macrophage infiltration and accumulation in the atherosclerotic lesion are associated with plaque progression and instability. Depletion of macrophages from the lesion might provide valuable insights into plaque stabilization processes. Therefore,...
2.
Lu C, Donners M, de Baaij J, Jin H, Otten J, Manca M, et al.
Front Immunol
. 2024 Feb;
15:1286382.
PMID: 38410507
Introduction: The impact of cardiovascular disease (CVD) risk factors, encompassing various biological determinants and unhealthy lifestyles, on the functional dynamics of circulating monocytes-a pivotal cell type in CVD pathophysiology remains...
3.
Rademakers T, van der Vorst E, Daissormont I, Otten J, Theodorou K, Theelen T, et al.
Sci Rep
. 2017 Mar;
7:45263.
PMID: 28349940
During plaque progression, inflammatory cells progressively accumulate in the adventitia, paralleled by an increased presence of leaky vasa vasorum. We here show that next to vasa vasorum, also the adventitial...
4.
Skarpengland T, Holm S, Scheffler K, Gregersen I, Dahl T, Suganthan R, et al.
Sci Rep
. 2016 Jun;
6:28337.
PMID: 27328939
Increasing evidence suggests that oxidative DNA damage accumulates in atherosclerosis. Recently, we showed that a genetic variant in the human DNA repair enzyme NEIL3 was associated with increased risk of...
5.
Borissoff J, Otten J, Heeneman S, Leenders P, Van Oerle R, Soehnlein O, et al.
PLoS One
. 2013 Feb;
8(2):e55784.
PMID: 23409043
Background: Variations in the blood coagulation activity, determined genetically or by medication, may alter atherosclerotic plaque progression, by influencing pleiotropic effects of coagulation proteases. Published experimental studies have yielded contradictory...
6.
Otten J, de Jager S, Kavelaars A, Seijkens T, Bot I, Wijnands E, et al.
FASEB J
. 2012 Oct;
27(1):265-76.
PMID: 23047899
Leukocyte chemotaxis is deemed instrumental in initiation and progression of atherosclerosis. It is mediated by G-protein-coupled receptors (e.g., CCR2 and CCR5), the activity of which is controlled by G-protein-coupled receptor...