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Jeremy Daniel

Explore the profile of Jeremy Daniel including associated specialties, affiliations and a list of published articles. Areas
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Articles 19
Citations 489
Followers 0
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Recent Articles
11.
Daniel J
S D Med . 2018 Feb; 71(1):35-36. PMID: 29439303
No abstract available.
12.
Daniel J, Owen A
S D Med . 2017 Aug; No:13-16. PMID: 28817844
With the discovery of morphine in the early 1800s, substance abuse quickly followed. Next came the production of heroin and other synthetic opioids, along with increases in nonmedical use of...
13.
Daniel J
S D Med . 2015 Jul; 68(6):262-3. PMID: 26137727
No abstract available.
14.
Pan W, Tu H, Hsuchou H, Daniel J, Kastin A
J Mol Neurosci . 2007 Oct; 33(3):232-8. PMID: 17952632
Cooperativity among ingestive peptides reflects attempts by the body to finely control its weight. Urocortin, like leptin, is a potent suppressor of food intake, and they interact at the blood-brain...
15.
Pan W, Kastin A, Daniel J, Yu C, Baryshnikova L, von Bartheld C
J Neuroimmunol . 2007 Feb; 185(1-2):47-56. PMID: 17316829
Using small tags, we tracked the pathway of tumor necrosis factor (TNF)alpha across cerebral vascular endothelial cells. In cerebral microvessel derived RBE4 cells, (125)I-TNFalpha had rapid endocytosis within the first...
16.
Kim J, Daniel J, Espejo A, Lake A, Krishna M, Xia L, et al.
EMBO Rep . 2006 Jan; 7(4):397-403. PMID: 16415788
The post-translational modification of histones regulates many cellular processes, including transcription, replication and DNA repair. A large number of combinations of post-translational modifications are possible. This cipher is referred to...
17.
Lee J, Sayegh J, Daniel J, Clarke S, Bedford M
J Biol Chem . 2005 Jul; 280(38):32890-6. PMID: 16051612
Protein arginine methylation is a common post-translational modification that has been implicated in signal transduction, RNA processing, transcriptional regulation, and DNA repair. A search of the human genome for additional...
18.
Spyres L, Daniel J, Hensley A, Qadan M, Ortiz-Leduc W, Ballard J
Infect Immun . 2003 May; 71(6):3294-301. PMID: 12761111
Toxin B (TcdB), a major Clostridium difficile virulence factor, glucosylates and inactivates the small GTP-binding proteins Rho, Rac, and Cdc42. In the present study we provide evidence that enzymatically inactive...
19.
Qadan M, Ramsey M, Daniel J, Spyres L, Safiejko-Mroczka B, Ortiz-Leduc W, et al.
Cell Microbiol . 2002 Jul; 4(7):425-34. PMID: 12102688
Clostridium difficile toxin B (TcdB) inactivates the small GTPases Rho, Rac and Cdc42 during intoxication of mammalian cells. In the current work, we show that TcdB has the potential to...